CCL2 and CCL5 mediate leukocyte adhesion in experimental autoimmune encephalomyelitis—an intravital microscopy study

Experimental autoimmune encephalomyelitis (EAE) models multiple sclerosis (MS) and is characterized by marked mononuclear cell influx in the brain. Several studies have demonstrated a role for chemokines during EAE. It remains to be determined whether these mediators modulate EAE primarily by mediat...

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Veröffentlicht in:	Journal of neuroimmunology 2005-05, Vol.162 (1), p.122-129
Hauptverfasser:	dos Santos, Adriana Carvalho, Barsante, Michele Mendes, Esteves Arantes, Rosa Maria, Bernard, Claude C.A., Teixeira, Mauro Martins, Carvalho-Tavares, Juliana
Format:	Artikel
Sprache:	eng
Schlagworte:	Analysis of Variance Animals Antibodies - pharmacology Body Weight - drug effects Brain Cell Adhesion - drug effects Cell Adhesion - physiology Cell Movement - drug effects Cell Movement - physiology Chemokine CCL2 - immunology Chemokine CCL2 - metabolism Chemokine CCL5 - metabolism Chemokines Chemokines, CC - immunology Chemokines, CC - metabolism Encephalomyelitis, Autoimmune, Experimental - chemically induced Encephalomyelitis, Autoimmune, Experimental - metabolism Encephalomyelitis, Autoimmune, Experimental - pathology Encephalomyelitis, Autoimmune, Experimental - physiopathology Enzyme-Linked Immunosorbent Assay - methods Female Glycoproteins - metabolism Intravital microscopy Leukocyte adhesion Leukocytes - drug effects Leukocytes - metabolism Mice Mice, Inbred C57BL Microcirculation - pathology Myelin-Oligodendrocyte Glycoprotein Peptide Fragments - metabolism Time Factors
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