Dual repressive effect of angiotensin II-type 1 receptor blocker telmisartan on angiotensin II-induced and estradiol-induced uterine leiomyoma cell proliferation

BACKGROUND Although uterine leiomyomas or fibroids are the most common gynecological benign tumor and greatly affect reproductive health and well-being, the pathophysiology and epidemiology of uterine leiomyomas are poorly understood. Elevated blood pressure has an independent, positive association...

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Veröffentlicht in:	Human reproduction (Oxford) 2008-02, Vol.23 (2), p.440-446
Hauptverfasser:	Isobe, Aki, Takeda, Takashi, Sakata, Masahiro, Miyake, Asako, Yamamoto, Toshiya, Minekawa, Ryoko, Nishimoto, Fumihito, Oskamoto, Yoko, Walker, Cheryl Lyn, Kimura, Tadashi
Format:	Artikel
Sprache:	eng
Schlagworte:	angiotensin II Angiotensin II - pharmacology angiotensin II type 1 receptor blocker Angiotensin II Type 1 Receptor Blockers - pharmacology Animals Benzimidazoles - pharmacology Benzoates - pharmacology Biological and medical sciences Cell Line, Tumor Cell Proliferation - drug effects Estradiol - pharmacology Female Gynecology. Andrology. Obstetrics Leiomyoma - chemically induced Leiomyoma - metabolism Leiomyoma - pathology Medical sciences Rats Receptor, Angiotensin, Type 1 - metabolism Receptor, Angiotensin, Type 2 - metabolism Signal Transduction telmisartan uterine leiomyoma Uterine Neoplasms - chemically induced Uterine Neoplasms - metabolism Uterine Neoplasms - pathology
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