Lactobacillus acidophilus suppresses intestinal inflammation by inhibiting endoplasmic reticulum stress

Background and Aim Nuclear factor kappa B (NF‐κB) activation and endoplasmic reticulum (ER) stress signaling play significant roles in the pathogenesis of inflammatory bowel disease (IBD). Thus, we evaluated whether new therapeutic probiotics have anti‐colitic effects, and we investigated their mech...

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Veröffentlicht in:	Journal of gastroenterology and hepatology 2019-01, Vol.34 (1), p.178-185
Hauptverfasser:	Kim, Da Hye, Kim, Soochan, Lee, Jin Ha, Kim, Jae Hyeon, Che, Xiumei, Ma, Hyun Woo, Seo, Dong Hyuk, Kim, Tae II, Kim, Won Ho, Kim, Seung Won, Cheon, Jae Hee
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Colitis Colitis - chemically induced Colitis - immunology Colitis - pathology Colitis - therapy Colon Cytokines Dextran Dextran Sulfate Endoplasmic reticulum Endoplasmic Reticulum Stress Flow cytometry Gene flow Goblet Cells HT29 Cells Humans Inflammatory bowel disease Inflammatory bowel diseases Interleukin 1 Interleukin 10 Interleukin-10 - metabolism Intestinal Mucosa - immunology Intestinal Mucosa - pathology Intestine Lactobacillus acidophilus Life span Lymphocytes T Macrophages Male Mice NF-kappa B Nitric oxide Nitric Oxide - antagonists & inhibitors nuclear factor kappa B Pediococcus pentosaceus Peritoneal Cavity - cytology Peritoneum Polymerase chain reaction Probiotics Probiotics - pharmacology RAW 264.7 Cells Sodium Splenocytes T-Lymphocytes, Regulatory - immunology Th2 Cells - immunology Tumor Necrosis Factor-alpha - antagonists & inhibitors Tumor necrosis factor-α
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