Modification of cocaine-induced behavioral and neurochemical effects by serotonin1A receptor agonist/antagonist in mice

Administration of cocaine causes a locomotor stimulant effect and increases extracellular levels of serotonin (5‐HT) and dopamine (DA) in the brains of rodents. Previous studies show that 5‐HT1A receptor agonist and antagonist modify the cocaine‐induced behavioral and neurochemical effects in the ra...

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Veröffentlicht in:	Synapse (New York, N.Y.) N.Y.), 2006-12, Vol.60 (7), p.479-484
Hauptverfasser:	Nakamura, Shigeo, Ago, Yukio, Hayashi, Aiko, Itoh, Soichi, Kakuda, Michiya, Hashimoto, Hitoshi, Baba, Akemichi, Matsuda, Toshio
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Behavior, Animal - drug effects Behavior, Animal - physiology Brain - drug effects Brain - metabolism cocaine Cocaine - antagonists & inhibitors Cocaine - metabolism Cocaine-Related Disorders - drug therapy Cocaine-Related Disorders - metabolism Cocaine-Related Disorders - physiopathology Disease Models, Animal Dopamine - metabolism Dopamine Uptake Inhibitors - antagonists & inhibitors Dopamine Uptake Inhibitors - metabolism Dose-Response Relationship, Drug Drug Interactions - physiology Extracellular Fluid - drug effects Extracellular Fluid - metabolism locomotor activity Male Mice Motor Activity - drug effects Motor Activity - physiology mouse osemozotan Prefrontal Cortex - drug effects Prefrontal Cortex - metabolism Receptor, Serotonin, 5-HT1A - drug effects Receptor, Serotonin, 5-HT1A - metabolism serotonin (5-HT)1A receptor Serotonin - metabolism Serotonin Agents - pharmacology Serotonin Antagonists - pharmacology Serotonin Receptor Agonists - pharmacology Time Factors Up-Regulation - drug effects Up-Regulation - physiology WAY100635
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