The a-chemokine CXCL14 is up-regulated in the sciatic nerve of a mouse model of Charcot-Marie-Tooth disease type 1A and alters myelin gene expression in cultured Schwann cells

At present the pathogenesis of CMT1A neuropathy, caused by the overexpression of PMP22, has not yet been entirely understood. The PMP22-overexpressing C61 mutant mouse is a suitable animal model, which mimics the human CMT1A disorder. We observed that myelin gene expression in the sciatic nerve of t...

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Veröffentlicht in:	Neurobiology of disease 2009-03, Vol.33 (3), p.448-458
Hauptverfasser:	Barbaria, Elena M, Kohl, Bianca, Buhren, Bettina A, Hasenpusch-Theil, Kerstin, Kruse, Fabian, Kury, Patrick, Martini, Rudolf, Muller, Hans Werner
Format:	Artikel
Sprache:	eng
Online-Zugang:	Volltext
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