Staphylococcus aureus triggers a shift from influenza virus–induced apoptosis to necrotic cell death

Superinfections with Staphylococcus aureus are a major complication of influenza disease, causing excessive inflammation and tissue damage. This enhanced cell‐damaging effect is also observed in superinfected tissue cultures, leading to a strong decrease in overall cell viability. In our analysis of...

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Veröffentlicht in:	The FASEB journal 2018-05, Vol.32 (5), p.2779-2793
Hauptverfasser:	Van Krüchten, Andre, Wilden, Janine J., Niemann, Silke, Peters, Georg, Löffler, Bettina, Ludwig, Stephan, Ehrhardt, Christina
Format:	Artikel
Sprache:	eng
Schlagworte:	accessory gene regulator locus Animals Apoptosis - immunology Caspase 8 - immunology caspase‐8 Dogs Human Umbilical Vein Endothelial Cells - immunology Human Umbilical Vein Endothelial Cells - microbiology Human Umbilical Vein Endothelial Cells - pathology Human Umbilical Vein Endothelial Cells - virology Humans Influenza A Virus, H1N1 Subtype - immunology Influenza, Human - immunology Influenza, Human - microbiology Influenza, Human - pathology Madin Darby Canine Kidney Cells Necrosis signaling Staphylococcal Infections - immunology Staphylococcal Infections - pathology Staphylococcal Infections - virology Staphylococcus aureus - immunology superinfection
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