Antibacterial and anti-atrophic effects of a highly soluble, acid stable UDCA formula in Helicobacter pylori-induced gastritis

Helicobacter pylori is one of the main causes of atrophic gastritis and gastric carcinogenesis. Gastritis can also occur in the absence of H. pylori as a result of bile reflux suggesting the eradication of H. pylori by bile acids. However, the bile salts are unable to eradicate H. pylori due to thei...

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Veröffentlicht in:	Biochemical pharmacology 2008-06, Vol.75 (11), p.2135-2146
Hauptverfasser:	Thao, Tran Dang Hien, Ryu, Ho-Cheol, Yoo, Seo-Hong, Rhee, Dong-Kwon
Format:	Artikel
Sprache:	eng
Schlagworte:	Adenocarcinoma - drug therapy Animals Anti-Bacterial Agents - pharmacology Anti-Bacterial Agents - therapeutic use Apoptosis - drug effects Bacterial diseases Bacterial diseases of the digestive system and abdomen Biological and medical sciences Cell Line, Tumor Dosage Forms Free Radical Scavengers Gastritis, Atrophic - drug therapy Gastritis, Atrophic - microbiology Gene Expression Regulation - drug effects Helicobacter Infections - drug therapy Helicobacter Infections - microbiology Helicobacter pylori Helicobacter pylori - drug effects Helicobacter Pylori-induced atrophic gastritis Human bacterial diseases Humans Hydrogen Peroxide - toxicity Infectious diseases Malate Dehydrogenase - metabolism Male Medical sciences Membrane Potential, Mitochondrial - drug effects Mice Mice, Inbred C57BL Mitochondria Neutrophils - drug effects Pharmacology. Drug treatments Reactive Oxygen Species RNA, Messenger - genetics RNA, Messenger - metabolism Stomach Neoplasms - drug therapy Time Factors Ursodeoxycholic acid (UDCA) Ursodeoxycholic Acid - pharmacology Ursodeoxycholic Acid - therapeutic use
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