c-FOS and n-NOS reactive neurons in response to circulating Phoneutria nigriventer spider venom

Abstract Drugs and neurotoxins activate specific neural circuits by increasing or decreasing the formation and release of neurotransmitters, such as nitric oxide (NO), and by inducing immediate early genes, such as FOS. We have previously shown that Phoneutria nigriventer spider venom (PNV) impairs...

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Veröffentlicht in:	Brain research bulletin 2007-06, Vol.73 (1), p.114-126
Hauptverfasser:	da Cruz-Höfling, Maria Alice, Zago, Gabriela Mariotoni, Melo, Liana Lins, Rapôso, Catarina
Format:	Artikel
Sprache:	eng
Schlagworte:	Acute stress-related area Amygdala - cytology Amygdala - drug effects Amygdala - metabolism Animals Araneae Brain Brain Chemistry - drug effects Cerebral Cortex - cytology Cerebral Cortex - drug effects Cerebral Cortex - metabolism Convulsion Data Interpretation, Statistical Hippocampus - cytology Hippocampus - drug effects Hippocampus - metabolism Immunohistochemistry Injections, Intravenous Male Motor-related area Neurology Neuronal activation Neurons - enzymology Neurons - physiology Nitric oxide Nitric Oxide Synthase Type I - metabolism Periaqueductal Gray - cytology Periaqueductal Gray - drug effects Periaqueductal Gray - metabolism Phoneutria nigriventer Proto-Oncogene Proteins c-fos - metabolism Rats Rats, Wistar Spider Venoms - administration & dosage Spider Venoms - pharmacology
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