Reply: Could the Interplay Between Macrophages and Fibroblasts Drive Extracellular Matrix Dynamics in End-Stage Heart Failure?
Contrary to the author’s suggestion that there is “no extensive inflammation or fibrosis” observed in our study, our values were 20 to 30 times greater than normal tissue. By looking at the change in a single variable, hemodynamic loading, we sought to narrow our focus to uncover pathways that could...
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Veröffentlicht in: | Journal of the American College of Cardiology 2017-12, Vol.70 (22), p.2838-2839 |
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Format: | Artikel |
Sprache: | eng |
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