Different mechanisms account for extracellular-signal regulated kinase activation in distinct brain regions following global ischemia and reperfusion

Abstract Oxidative stress after cerebral ischemia and reperfusion activates extracellular signal-regulated kinases (ERK) in brain. However, the mechanism of this activation has not been elucidated. We have previously reported that in an in vitro model of oxidative stress in immature cortical neurona...

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Veröffentlicht in:Neuroscience 2007-03, Vol.145 (1), p.248-255
Hauptverfasser: Ho, Y, Logue, E, Callaway, C.W, DeFranco, D.B
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Sprache:eng
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