Status epilepticus induces region-specific changes in dendritic spines, dendritic length and TrkB protein content of rat brain cortex

Abstract Induction of status epilepticus (SE) with kainic acid results in a large reorganization of neuronal brain circuits, a phenomenon that has been studied primarily in the hippocampus. The neurotrophin BDNF, by acting through its receptor TrkB, has been implicated in such reorganization. In the...

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Veröffentlicht in:	Brain research 2007-05, Vol.1150, p.225-238
Hauptverfasser:	Ampuero, Estíbaliz, Dagnino-Subiabre, Alexies, Sandoval, Rodrigo, Zepeda-Carreño, Rodrigo, Sandoval, Soledad, Viedma, Alejandra, Aboitiz, Francisco, Orrego, Fernando, Wyneken, Ursula
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Apoptosis Biological and medical sciences Cerebral Cortex - pathology Dendritic morphology Dendritic Spines - pathology Dendritic Spines - ultrastructure Disease Models, Animal Headache. Facial pains. Syncopes. Epilepsia. Intracranial hypertension. Brain oedema. Cerebral palsy In Situ Nick-End Labeling - methods Kainic Acid Male Medical sciences Neocortex Nervous system (semeiology, syndromes) Neurology Neurons - pathology Rats Rats, Sprague-Dawley Receptor, trkB - metabolism Seizure Silver Staining - methods Statistics, Nonparametric Status Epilepticus - chemically induced Status Epilepticus - metabolism Status Epilepticus - pathology Time Factors TrkB
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