Normal pancreatic β-cell function in mice with RIP-Cre-mediated inactivation of p62/SQSTM1

Recent studies have suggested that decreased pancreatic β-cell function and mass are common features of patients with type 2 diabetes mellitus. Pancreatic β-cell homeostasis is regulated by various types of signaling molecules and stress responses. Sequestosome 1/p62 (SQSTM1, hereafter referred to a...

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Veröffentlicht in:	Endocrine Journal 2018, Vol.65(1), pp.83-89
Hauptverfasser:	Honda, Akira, Komiya, Koji, Hara, Akemi, Fukunaka, Ayako, Suzuki, Luka, Miyatsuka, Takeshi, Ogihara, Takeshi, Fujitani, Yoshio, Watada, Hirotaka
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Autophagy Blood Glucose - analysis Cell Proliferation Crosses, Genetic Diabetes Gene Expression Immunohistochemistry Insulin - blood Insulin - genetics Insulin - metabolism Insulin Secretion Insulin-Secreting Cells - cytology Insulin-Secreting Cells - metabolism Islets Islets of Langerhans - cytology Islets of Langerhans - metabolism Male Mice, Knockout Mice, Transgenic Organ Specificity p62 Promoter Regions, Genetic Reproducibility of Results RNA, Messenger - metabolism Sequestosome 1 Sequestosome-1 Protein - antagonists & inhibitors Sequestosome-1 Protein - genetics Sequestosome-1 Protein - metabolism Specific Pathogen-Free Organisms Weight Gain β cell
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