MiR-93-5p inhibits the EMT of breast cancer cells via targeting MKL-1 and STAT3

Epithelial-mesenchymal transition (EMT) plays an important role in breast cancer cell metastasis. Both (megakaryoblastic leukemia)/myocardin-like 1 (MKL-1) and Signal transducer and activator of transcription 3 (STAT3) have been implicated in the control of cellular metabolism, survival and growth....

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Veröffentlicht in:	Experimental cell research 2017-08, Vol.357 (1), p.135-144
Hauptverfasser:	Xiang, Yuan, Liao, Xing-Hua, Yu, Cheng-Xi, Yao, Ao, Qin, Huan, Li, Jia-Peng, Hu, Peng, Li, Hui, Guo, Wei, Gu, Chao-Jiang, Zhang, Tong-Cun
Format:	Artikel
Sprache:	eng
Schlagworte:	Breast Neoplasms - genetics Breast Neoplasms - metabolism EMT Epithelial-Mesenchymal Transition - physiology Gene Expression Regulation, Neoplastic - genetics Humans MCF-7 Cells MicroRNAs - genetics miR-93-5p MKL-1 Promoter Regions, Genetic - genetics STAT3 STAT3 Transcription Factor - genetics The migration breast cancer cells Trans-Activators - genetics
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