Kisspeptin in the Hypothalamus of 2 Rat Models of Polycystic Ovary Syndrome

Abstract Hyperandrogenism, disturbance of the hypothalamus-pituitary-ovary axis followed by elevated serum luteinizing hormone (LH) levels, and insulin resistance are involved in the complicated pathophysiology of polycystic ovary syndrome (PCOS). Kisspeptin is coexpressed with neurokinin B (NKB) in...

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Veröffentlicht in:	Endocrinology (Philadelphia) 2017-02, Vol.158 (2), p.367-377
Hauptverfasser:	Osuka, Satoko, Iwase, Akira, Nakahara, Tatsuo, Kondo, Mika, Saito, Ai, Bayasula, Nakamura, Tomoko, Takikawa, Sachiko, Goto, Maki, Kotani, Tomomi, Kikkawa, Fumitaka
Format:	Artikel
Sprache:	eng
Schlagworte:	Androgens Animal models Animals Arcuate nucleus Arcuate Nucleus of Hypothalamus - metabolism Body Weight Body weight gain Dihydrotestosterone Disease Models, Animal Endocrinology Estrous Cycle Estrus cycle Female Gonadal Steroid Hormones - blood Gonadotropin-releasing hormone Gonadotropins Gonadotropins - blood Hypothalamo-Hypophyseal System - metabolism Hypothalamus Hypothalamus, Anterior - metabolism Insulin Insulin resistance Kiss1 protein Kisspeptins - metabolism Luteinizing hormone Neurokinin Neurokinin B Ovaries Ovary - metabolism Ovary - pathology Phenotype Phenotypes Pituitary Pituitary (anterior) Polycystic ovary syndrome Polycystic Ovary Syndrome - metabolism Polycystic Ovary Syndrome - pathology Prenatal experience Prenatal exposure Pulse generators Rats Rats, Wistar Rodents Secretion Serum levels Weight
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