Enhancement of Nitric Oxide Production Is Responsible for Minimal Intimal Hyperplasia of Autogenous Rabbit Arterial Grafts

Background:Vascular endothelium induces smooth muscle cell (SMC) relaxation mainly mediated by endothelium-derived nitric oxide (EDNO) and endothelium-derived hyperpolarizing factor (EDHF). It has previously been reported that functions of these endothelium factors have been greatly impaired in vein...

Ausführliche Beschreibung

Gespeichert in:

Bibliographische Detailangaben
Veröffentlicht in:	Circulation Journal 2017/07/25, Vol.81(8), pp.1222-1230
Hauptverfasser:	Tabata, Koki, Komori, Kimihiro, Otsuka, Ryo, Kajikuri, Junko, Itoh, Takeo
Format:	Artikel
Sprache:	eng
Schlagworte:	Acetylcholine Animals Autografts Biological Factors - metabolism Calcium - metabolism Calcium-activated K+ channels Carotid Arteries - metabolism Carotid Arteries - pathology Carotid Arteries - transplantation Endothelial cell Ca2+ concentration Endothelium, Vascular - metabolism Endothelium, Vascular - pathology Endothelium-dependent smooth muscle cell hyperpolarization Hyperplasia - etiology Hyperplasia - metabolism Hyperplasia - pathology Nitric oxide Nitric Oxide - metabolism Rabbits Tunica Media - metabolism Tunica Media - pathology
Online-Zugang:	Volltext
Tags:	Tag hinzufügen Keine Tags, Fügen Sie den ersten Tag hinzu!

Schreiben Sie den ersten Kommentar!