Suppression of uterine decidualization correlated with reduction in serum progesterone levels as a cause of preimplantation embryonic loss induced by diphenyltin in rats

In our previous study, diphenyltin dichloride (DPTCl) at 16.5 mg/kg and higher on days 0–3 of pregnancy was found to induce preimplantation embryonic loss in rats. In the present study, the effects of DPTCl on uterine decidualization in pseudopregnant rats, effects of ovarian hormones on uterine dec...

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Veröffentlicht in:	Reproductive toxicology (Elmsford, N.Y.) N.Y.), 2002-05, Vol.16 (3), p.309-317
Hauptverfasser:	Ema, Makoto, Miyawaki, Emiko
Format:	Artikel
Sprache:	eng
Schlagworte:	Administration, Oral Animals Biological and medical sciences Blastocyst - drug effects Blastocyst - pathology Chemical and industrial products toxicology. Toxic occupational diseases Decidua - drug effects Decidua - pathology Decidual cell response Diphenyltin Dose-Response Relationship, Drug Embryo Implantation - drug effects Environmental Pollutants - administration & dosage Environmental Pollutants - toxicity Estrone - pharmacology Female Fertility - drug effects Male Medical sciences Metals and various inorganic compounds Organ Size - drug effects Organotin Compounds - administration & dosage Organotin Compounds - toxicity Ovariectomy Ovary - drug effects Ovary - pathology Pregnancy Preimplantation embryonic loss Progesterone Progesterone - blood Progesterone - pharmacology Pseudopregnancy Rats Rats, Inbred WKY Toxicology
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