Contrast Media-Induced Renal Inflammation Is Mediated Through HMGB1 and Its Receptors in Human Tubular Cells

With the rapid development of imaging diagnosis and interventional therapy, contrast media (CM) are widely used in clinics. However, contrast-induced nephropathy (CIN) is the third leading cause of hospital-acquired acute renal failure accounting for 10-12% of all causes of hospital-acquired renal f...

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Veröffentlicht in:	DNA and cell biology 2017-01, Vol.36 (1), p.67-76
Hauptverfasser:	Guan, Xiao-Feng, Chen, Qing-Jie, Zuo, Xiao-Cong, Guo, Ren, Peng, Xiang-Dong, Wang, Jiang-Lin, Yin, Wen-Jun, Li, Dai-Yang
Format:	Artikel
Sprache:	eng
Schlagworte:	Active Transport, Cell Nucleus - drug effects Cell Line Cell Nucleus - drug effects Cell Nucleus - metabolism Chemokine CCL2 - secretion Contrast Media - pharmacology Diatrizoate Meglumine - pharmacology Epithelial Cells - metabolism Epithelial Cells - secretion Gene Expression Regulation - drug effects Glycyrrhizic Acid - pharmacology HMGB1 Protein - metabolism Humans Inflammation - chemically induced Inflammation - metabolism Inflammation - pathology Interleukin-6 - secretion Kidney Tubules - drug effects Kidney Tubules - metabolism Kidney Tubules - pathology Necrosis - chemically induced Necrosis - metabolism Receptors, CXCR4 - metabolism Signal Transduction - drug effects Toll-Like Receptor 2 - metabolism Transcription Factor RelA - metabolism
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