Differences in Aβ brain networks in Alzheimer's disease and healthy controls

Abstract The prevailing β-amyloid (Aβ)-cascade hypothesis is the most classical Alzheimer's disease (AD) pathogenesis. In this hypothesis, excessive Aβ plaque deposition in human brain is considered to be the cause of AD. Carbon 11-labeled Pittsburgh compound B Positron emission tomography (11C...

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Veröffentlicht in:	Brain research 2017-01, Vol.1655, p.77-89
Hauptverfasser:	Duan, Huoqiang, Jiang, Jiehui, Xu, Jun, Zhou, Hucheng, Huang, Zhemin, Yu, Zhihua, Yan, Zhuangzhi
Format:	Artikel
Sprache:	eng
Schlagworte:	11C-PiB PET Age Factors Aged Alzheimer Disease - diagnostic imaging Alzheimer Disease - metabolism Alzheimer's disease Amyloid beta-Peptides - metabolism Benzothiazoles Body Weight Brain - diagnostic imaging Brain - metabolism Brain Mapping Brain networks Cluster Analysis Datasets as Topic Female Graph theoretical Humans Male Neural Pathways - diagnostic imaging Neural Pathways - metabolism Neurology Positron-Emission Tomography Radiopharmaceuticals Regression Analysis ROC Curve Sex Factors Signal Processing, Computer-Assisted β-amyloid (Aβ)-cascade
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