Systemic administration of anorexic gut peptide hormones impairs hedonic-driven sucrose consumption in mice

Abstract A number of reports suggest that gut hormones such as cholecystokinin (CCK), glucagon-like peptide 1 (GLP-1), and peptide YY (3 – 36) (PYY3 – 36 ), which are released postprandially, suppress homeostatic food intake and result in satiety and the termination of feeding. However, it remains u...

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Veröffentlicht in:	Physiology & behavior 2017-03, Vol.171, p.158-164
Hauptverfasser:	Yamaguchi, Erina, Yasoshima, Yasunobu, Shimura, Tsuyoshi
Format:	Artikel
Sprache:	eng
Schlagworte:	Analysis of Variance Animals Antimanic Agents - pharmacology Avoidance Learning - drug effects Cholecystokinin Conditioned taste aversion Disease Models, Animal Dose-Response Relationship, Drug Eating - drug effects Gastrointestinal Hormones - pharmacology Glucagon-like peptide 1 Glucagon-Like Peptide 1 - pharmacology Hedonic-driven consumption Lithium Chloride - pharmacology Male Mice Mice, Inbred C57BL Peptide Fragments - pharmacology Peptide YY - pharmacology Peptide YY(3–36) Philosophy Psychiatry Sincalide - pharmacology Sucrose Sucrose - administration & dosage Sweetening Agents - administration & dosage Taste - drug effects Time Factors
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