Hypothalamic-Pituitary-Adrenal Hypofunction in Myalgic Encephalomyelitis (ME)/Chronic Fatigue Syndrome (CFS) as a Consequence of Activated Immune-Inflammatory and Oxidative and Nitrosative Pathways

There is evidence that immune-inflammatory and oxidative and nitrosative stress (O&NS) pathways play a role in the pathophysiology of myalgic encephalomyelitis (ME)/chronic fatigue syndrome (CFS). There is also evidence that these neuroimmune diseases are accompanied by hypothalamic-pituitary-ad...

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Veröffentlicht in:	Molecular neurobiology 2017-11, Vol.54 (9), p.6806-6819
Hauptverfasser:	Morris, Gerwyn, Anderson, George, Maes, Michael
Format:	Artikel
Sprache:	eng
Schlagworte:	Activation Adrenal glands Adrenocorticotropic hormone Animals Bacteria Bidirectional Biomedical and Life Sciences Biomedicine Cell Biology Chronic fatigue syndrome Cortisol Encephalomyelitis Fatigue Fatigue Syndrome, Chronic - immunology Fatigue Syndrome, Chronic - metabolism Feedback Glucocorticoids Humans Hypothalamic-pituitary-adrenal axis Hypothalamo-Hypophyseal System - immunology Hypothalamo-Hypophyseal System - metabolism Hypothalamus Hypothalamus - immunology Hypothalamus - metabolism Immune system Immunity, Cellular - physiology Inflammation Inflammation - immunology Inflammation - metabolism Inflammation Mediators - immunology Inflammation Mediators - metabolism Inflammatory diseases Interleukin 10 Negative feedback Neurobiology Neurology Neurosciences Nitric oxide Nitrosation - physiology Oxidative stress Oxidative Stress - physiology Pituitary Pituitary gland Pituitary-Adrenal System - immunology Pituitary-Adrenal System - metabolism Signal Transduction - physiology Transforming growth factor Transforming growth factor-b Tumor necrosis factor-α
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