Genetic association between RAGE polymorphisms and Alzheimer's disease and Lewy body dementias in a Japanese cohort: a case–control study

Background/Aims Interaction of receptor for advanced glycation end products (RAGE) with amyloid‐β increases amplification of oxidative stress and plays pathological roles in Alzheimer's disease (AD). Oxidative stress leads to α‐synuclein aggregation and is also a major contributing factor in th...

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Veröffentlicht in:	International journal of geriatric psychiatry 2017-12, Vol.32 (12), p.1241-1246
Hauptverfasser:	Takeshita, Yoshihide, Shibata, Nobuto, Kasanuki, Koji, Nagata, Tomoyuki, Shinagawa, Shunichiro, Kobayashi, Nobuyuki, Ohnuma, Tohru, Suzuki, Ayako, Kawai, Eri, Takayama, Toshiki, Nishioka, Kenya, Motoi, Yumiko, Hattori, Nobutaka, Nakayama, Kazuhiko, Yamada, Hisashi, Arai, Heii
Format:	Artikel
Sprache:	eng
Schlagworte:	Advanced glycosylation end products Aged Aged, 80 and over Alzheimer Disease - genetics Alzheimer's disease apolipoprotein E Case-Control Studies Cohort Studies Dementia Female Genetic Predisposition to Disease Geriatric psychiatry Glycosylation Haplotypes Humans Japan Lewy bodies Lewy body dementias Lewy Body Disease - genetics Linkage Disequilibrium Male Middle Aged Neurodegenerative diseases Oxidative stress Polymorphism Polymorphism, Single Nucleotide Receptor for Advanced Glycation End Products - genetics receptor for advanced glycation end‐products (RAGE) Receptors, Immunologic Risk Single-nucleotide polymorphism Synuclein
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