Inflammatory and neuropathic cold allodynia are selectively mediated by the neurotrophic factor receptor GFR alpha 3

Tissue injury prompts the release of a number of proalgesic molecules that induce acute and chronic pain by sensitizing pain-sensing neurons (nociceptors) to heat and mechanical stimuli. In contrast, many proalgesics have no effect on cold sensitivity or can inhibit cold-sensitive neurons and dimini...

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Veröffentlicht in:	Proceedings of the National Academy of Sciences - PNAS 2016-04, Vol.113 (16), p.4506-4506
Hauptverfasser:	Lippoldt, Erika K, Ongun, Serra, Kusaka, Geoffrey K, McKemy, David D
Format:	Artikel
Sprache:	eng
Online-Zugang:	Volltext
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