Mitochondrial c-Fos May Increase the Vulnerability of Neuro2a Cells to Cellular Stressors

Although c-Fos expression in mitochondria is known to increase under excitatory injury via kainic acid or N -methyl- d -aspartate injection, the authentic function of c-Fos in mitochondria remains unknown. We found that c-Fos expression in the mitochondria of neuroblastoma Neuro2a cells was augmente...

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Veröffentlicht in:	Journal of molecular neuroscience 2016-05, Vol.59 (1), p.106-112
Hauptverfasser:	Kambe, Yuki, Miyata, Atsuro
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Biomedical and Life Sciences Biomedicine Cell Biology Cell Hypoxia Cell Line, Tumor Cytochrome Glucose Glucose - deficiency Glucose - metabolism Ischemia Mice Mitochondria Mitochondria - metabolism Mitochondrial DNA Neurochemistry Neurology Neurons - drug effects Neurons - metabolism Neurosciences Oxidative Stress Polymerase chain reaction Proteomics Proto-Oncogene Proteins c-fos - genetics Proto-Oncogene Proteins c-fos - metabolism Rotenone - toxicity Sodium
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