Traumatic brain injury and obesity induce persistent central insulin resistance

Traumatic brain injury (TBI)‐induced impairments in cerebral energy metabolism impede tissue repair and contribute to delayed functional recovery. Moreover, the transient alteration in brain glucose utilization corresponds to a period of increased vulnerability to the negative effects of a subsequen...

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Veröffentlicht in:	The European journal of neuroscience 2016-04, Vol.43 (8), p.1034-1043
Hauptverfasser:	Karelina, Kate, Sarac, Benjamin, Freeman, Lindsey M., Gaier, Kristopher R., Weil, Zachary M.
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals anxiety Brain - metabolism Brain Injuries, Traumatic - complications Brain Injuries, Traumatic - metabolism Brain Injuries, Traumatic - physiopathology central metabolism high-fat diet Insulin - metabolism insulin receptor Insulin Resistance Learning Mice Mice, Inbred C57BL mouse neuroinflammation Obesity - complications Obesity - metabolism Obesity - physiopathology Proto-Oncogene Proteins c-akt - metabolism Signal Transduction
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