Conditional Granger causality and partitioned Granger causality: differences and similarities

Neural information modeling and analysis often requires a measurement of the mutual influence among many signals. A common technique is the conditional Granger causality (cGC) which measures the influence of one time series on another time series in the presence of a third. Geweke has translated thi...

Ausführliche Beschreibung

Gespeichert in:
Bibliographische Detailangaben
Veröffentlicht in:Biological cybernetics 2015-12, Vol.109 (6), p.627-637
Hauptverfasser: Malekpour, Sheida, Sethares, William A.
Format: Artikel
Sprache:eng
Schlagworte:
Online-Zugang:Volltext
Tags: Tag hinzufügen
Keine Tags, Fügen Sie den ersten Tag hinzu!
container_end_page 637
container_issue 6
container_start_page 627
container_title Biological cybernetics
container_volume 109
creator Malekpour, Sheida
Sethares, William A.
description Neural information modeling and analysis often requires a measurement of the mutual influence among many signals. A common technique is the conditional Granger causality (cGC) which measures the influence of one time series on another time series in the presence of a third. Geweke has translated this condition into the frequency domain and has explored the mathematical relationships between the time and frequency domain expressions. Chen has observed that in practice, the expressions may return (meaningless) negative numbers, and has proposed an alternative which is based on a partitioned matrix scheme, which we call partitioned Granger causality (pGC). There has been some confusion in the literature about the relationship between cGC and pGC; some authors treat them as essentially identical measures, while others have noted that some properties (such as the relationship between the time and frequency domain expressions) do not hold for the pGC. This paper presents a series of matrix equalities that simplify the calculation of the pGC. In this simplified expression, the essential differences and similarities between the cGC and the pGC become clear; in essence, the pGC is dependent on only a subset of the parameters in the model estimation, and the noise residuals (which are uncorrelated in the cGC) need not be uncorrelated in the pGC. The mathematical results are illustrated with a simulation, and the measures are applied to an EEG dataset.
doi_str_mv 10.1007/s00422-015-0665-3
format Article
fullrecord <record><control><sourceid>proquest_cross</sourceid><recordid>TN_cdi_proquest_miscellaneous_1777997682</recordid><sourceformat>XML</sourceformat><sourcesystem>PC</sourcesystem><sourcerecordid>3874576991</sourcerecordid><originalsourceid>FETCH-LOGICAL-c508t-a51f92bf417fa491aeeb76d08498431986ed8e4396074ebcf41a13a6d796c0b23</originalsourceid><addsrcrecordid>eNqNkU1LxDAURYMozvjxA9xIwY2b6st3404GHQXBjS4lpO2rVDrtmLSL-femMyqiCLMKJOfePN4h5ITCBQXQlwFAMJYClSkoJVO-Q6ZU8HijNeySKXABKWUAE3IQwhsAGCbNPpkwJbTItJqSl1nXlnVfd61rkrl37Sv6pHBDcE3drxLXlsnS-X5NYPmXuErKuqrQY1tgWOOhXtSN8zGB4YjsVa4JePx5HpLn25un2V368Di_n10_pIWErE-dpJVheSWorpww1CHmWpWQCZMJTk2msMxQcKNAC8yLCDrKnSq1UQXkjB-S803v0nfvA4beLupQYNO4FrshWKq1NkarbBs07gZoJs0WqKSMxSF1RM9-oW_d4ONOR4pLaZSiYyHdUIXvQvBY2aWvF86vLAU7GrUbozYataNRy2Pm9LN5yBdYfie-FEaAbYAQn0Y5P77-t_UDBnOqlA</addsrcrecordid><sourcetype>Aggregation Database</sourcetype><iscdi>true</iscdi><recordtype>article</recordtype><pqid>1735596619</pqid></control><display><type>article</type><title>Conditional Granger causality and partitioned Granger causality: differences and similarities</title><source>MEDLINE</source><source>SpringerLink Journals - AutoHoldings</source><creator>Malekpour, Sheida ; Sethares, William A.</creator><creatorcontrib>Malekpour, Sheida ; Sethares, William A.</creatorcontrib><description>Neural information modeling and analysis often requires a measurement of the mutual influence among many signals. A common technique is the conditional Granger causality (cGC) which measures the influence of one time series on another time series in the presence of a third. Geweke has translated this condition into the frequency domain and has explored the mathematical relationships between the time and frequency domain expressions. Chen has observed that in practice, the expressions may return (meaningless) negative numbers, and has proposed an alternative which is based on a partitioned matrix scheme, which we call partitioned Granger causality (pGC). There has been some confusion in the literature about the relationship between cGC and pGC; some authors treat them as essentially identical measures, while others have noted that some properties (such as the relationship between the time and frequency domain expressions) do not hold for the pGC. This paper presents a series of matrix equalities that simplify the calculation of the pGC. In this simplified expression, the essential differences and similarities between the cGC and the pGC become clear; in essence, the pGC is dependent on only a subset of the parameters in the model estimation, and the noise residuals (which are uncorrelated in the cGC) need not be uncorrelated in the pGC. The mathematical results are illustrated with a simulation, and the measures are applied to an EEG dataset.</description><identifier>ISSN: 0340-1200</identifier><identifier>EISSN: 1432-0770</identifier><identifier>DOI: 10.1007/s00422-015-0665-3</identifier><identifier>PMID: 26474876</identifier><language>eng</language><publisher>Berlin/Heidelberg: Springer Berlin Heidelberg</publisher><subject>Analogies ; Bioinformatics ; Biomedical and Life Sciences ; Biomedicine ; Causality ; Complex Systems ; Computer Appl. in Life Sciences ; Computer simulation ; Confusion ; Cybernetics ; Datasets ; Electroencephalography ; Frequency domains ; Mathematical models ; Models, Theoretical ; Neurobiology ; Neurology ; Neurosciences ; Noise ; Original Article ; Time series</subject><ispartof>Biological cybernetics, 2015-12, Vol.109 (6), p.627-637</ispartof><rights>Springer-Verlag Berlin Heidelberg 2015</rights><lds50>peer_reviewed</lds50><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c508t-a51f92bf417fa491aeeb76d08498431986ed8e4396074ebcf41a13a6d796c0b23</citedby><cites>FETCH-LOGICAL-c508t-a51f92bf417fa491aeeb76d08498431986ed8e4396074ebcf41a13a6d796c0b23</cites></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><linktopdf>$$Uhttps://link.springer.com/content/pdf/10.1007/s00422-015-0665-3$$EPDF$$P50$$Gspringer$$H</linktopdf><linktohtml>$$Uhttps://link.springer.com/10.1007/s00422-015-0665-3$$EHTML$$P50$$Gspringer$$H</linktohtml><link.rule.ids>314,780,784,27924,27925,41488,42557,51319</link.rule.ids><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/26474876$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Malekpour, Sheida</creatorcontrib><creatorcontrib>Sethares, William A.</creatorcontrib><title>Conditional Granger causality and partitioned Granger causality: differences and similarities</title><title>Biological cybernetics</title><addtitle>Biol Cybern</addtitle><addtitle>Biol Cybern</addtitle><description>Neural information modeling and analysis often requires a measurement of the mutual influence among many signals. A common technique is the conditional Granger causality (cGC) which measures the influence of one time series on another time series in the presence of a third. Geweke has translated this condition into the frequency domain and has explored the mathematical relationships between the time and frequency domain expressions. Chen has observed that in practice, the expressions may return (meaningless) negative numbers, and has proposed an alternative which is based on a partitioned matrix scheme, which we call partitioned Granger causality (pGC). There has been some confusion in the literature about the relationship between cGC and pGC; some authors treat them as essentially identical measures, while others have noted that some properties (such as the relationship between the time and frequency domain expressions) do not hold for the pGC. This paper presents a series of matrix equalities that simplify the calculation of the pGC. In this simplified expression, the essential differences and similarities between the cGC and the pGC become clear; in essence, the pGC is dependent on only a subset of the parameters in the model estimation, and the noise residuals (which are uncorrelated in the cGC) need not be uncorrelated in the pGC. The mathematical results are illustrated with a simulation, and the measures are applied to an EEG dataset.</description><subject>Analogies</subject><subject>Bioinformatics</subject><subject>Biomedical and Life Sciences</subject><subject>Biomedicine</subject><subject>Causality</subject><subject>Complex Systems</subject><subject>Computer Appl. in Life Sciences</subject><subject>Computer simulation</subject><subject>Confusion</subject><subject>Cybernetics</subject><subject>Datasets</subject><subject>Electroencephalography</subject><subject>Frequency domains</subject><subject>Mathematical models</subject><subject>Models, Theoretical</subject><subject>Neurobiology</subject><subject>Neurology</subject><subject>Neurosciences</subject><subject>Noise</subject><subject>Original Article</subject><subject>Time series</subject><issn>0340-1200</issn><issn>1432-0770</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2015</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><sourceid>ABUWG</sourceid><sourceid>AFKRA</sourceid><sourceid>AZQEC</sourceid><sourceid>BENPR</sourceid><sourceid>CCPQU</sourceid><sourceid>DWQXO</sourceid><sourceid>GNUQQ</sourceid><recordid>eNqNkU1LxDAURYMozvjxA9xIwY2b6st3404GHQXBjS4lpO2rVDrtmLSL-femMyqiCLMKJOfePN4h5ITCBQXQlwFAMJYClSkoJVO-Q6ZU8HijNeySKXABKWUAE3IQwhsAGCbNPpkwJbTItJqSl1nXlnVfd61rkrl37Sv6pHBDcE3drxLXlsnS-X5NYPmXuErKuqrQY1tgWOOhXtSN8zGB4YjsVa4JePx5HpLn25un2V368Di_n10_pIWErE-dpJVheSWorpww1CHmWpWQCZMJTk2msMxQcKNAC8yLCDrKnSq1UQXkjB-S803v0nfvA4beLupQYNO4FrshWKq1NkarbBs07gZoJs0WqKSMxSF1RM9-oW_d4ONOR4pLaZSiYyHdUIXvQvBY2aWvF86vLAU7GrUbozYataNRy2Pm9LN5yBdYfie-FEaAbYAQn0Y5P77-t_UDBnOqlA</recordid><startdate>20151201</startdate><enddate>20151201</enddate><creator>Malekpour, Sheida</creator><creator>Sethares, William A.</creator><general>Springer Berlin Heidelberg</general><general>Springer Nature B.V</general><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>3V.</scope><scope>7QO</scope><scope>7TK</scope><scope>7X7</scope><scope>7XB</scope><scope>88A</scope><scope>88E</scope><scope>88I</scope><scope>8AL</scope><scope>8AO</scope><scope>8FD</scope><scope>8FE</scope><scope>8FG</scope><scope>8FH</scope><scope>8FI</scope><scope>8FJ</scope><scope>8FK</scope><scope>ABUWG</scope><scope>AFKRA</scope><scope>ARAPS</scope><scope>AZQEC</scope><scope>BBNVY</scope><scope>BENPR</scope><scope>BGLVJ</scope><scope>BHPHI</scope><scope>CCPQU</scope><scope>DWQXO</scope><scope>FR3</scope><scope>FYUFA</scope><scope>GHDGH</scope><scope>GNUQQ</scope><scope>H8D</scope><scope>HCIFZ</scope><scope>JQ2</scope><scope>K7-</scope><scope>K9.</scope><scope>L7M</scope><scope>LK8</scope><scope>M0N</scope><scope>M0S</scope><scope>M1P</scope><scope>M2P</scope><scope>M7P</scope><scope>P5Z</scope><scope>P62</scope><scope>P64</scope><scope>PQEST</scope><scope>PQQKQ</scope><scope>PQUKI</scope><scope>Q9U</scope><scope>7X8</scope></search><sort><creationdate>20151201</creationdate><title>Conditional Granger causality and partitioned Granger causality: differences and similarities</title><author>Malekpour, Sheida ; Sethares, William A.</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c508t-a51f92bf417fa491aeeb76d08498431986ed8e4396074ebcf41a13a6d796c0b23</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2015</creationdate><topic>Analogies</topic><topic>Bioinformatics</topic><topic>Biomedical and Life Sciences</topic><topic>Biomedicine</topic><topic>Causality</topic><topic>Complex Systems</topic><topic>Computer Appl. in Life Sciences</topic><topic>Computer simulation</topic><topic>Confusion</topic><topic>Cybernetics</topic><topic>Datasets</topic><topic>Electroencephalography</topic><topic>Frequency domains</topic><topic>Mathematical models</topic><topic>Models, Theoretical</topic><topic>Neurobiology</topic><topic>Neurology</topic><topic>Neurosciences</topic><topic>Noise</topic><topic>Original Article</topic><topic>Time series</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Malekpour, Sheida</creatorcontrib><creatorcontrib>Sethares, William A.</creatorcontrib><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>ProQuest Central (Corporate)</collection><collection>Biotechnology Research Abstracts</collection><collection>Neurosciences Abstracts</collection><collection>Health &amp; Medical Collection</collection><collection>ProQuest Central (purchase pre-March 2016)</collection><collection>Biology Database (Alumni Edition)</collection><collection>Medical Database (Alumni Edition)</collection><collection>Science Database (Alumni Edition)</collection><collection>Computing Database (Alumni Edition)</collection><collection>ProQuest Pharma Collection</collection><collection>Technology Research Database</collection><collection>ProQuest SciTech Collection</collection><collection>ProQuest Technology Collection</collection><collection>ProQuest Natural Science Collection</collection><collection>Hospital Premium Collection</collection><collection>Hospital Premium Collection (Alumni Edition)</collection><collection>ProQuest Central (Alumni) (purchase pre-March 2016)</collection><collection>ProQuest Central (Alumni Edition)</collection><collection>ProQuest Central UK/Ireland</collection><collection>Advanced Technologies &amp; Aerospace Collection</collection><collection>ProQuest Central Essentials</collection><collection>Biological Science Collection</collection><collection>ProQuest Central</collection><collection>Technology Collection</collection><collection>Natural Science Collection</collection><collection>ProQuest One Community College</collection><collection>ProQuest Central Korea</collection><collection>Engineering Research Database</collection><collection>Health Research Premium Collection</collection><collection>Health Research Premium Collection (Alumni)</collection><collection>ProQuest Central Student</collection><collection>Aerospace Database</collection><collection>SciTech Premium Collection</collection><collection>ProQuest Computer Science Collection</collection><collection>Computer Science Database</collection><collection>ProQuest Health &amp; Medical Complete (Alumni)</collection><collection>Advanced Technologies Database with Aerospace</collection><collection>ProQuest Biological Science Collection</collection><collection>Computing Database</collection><collection>Health &amp; Medical Collection (Alumni Edition)</collection><collection>Medical Database</collection><collection>Science Database</collection><collection>Biological Science Database</collection><collection>Advanced Technologies &amp; Aerospace Database</collection><collection>ProQuest Advanced Technologies &amp; Aerospace Collection</collection><collection>Biotechnology and BioEngineering Abstracts</collection><collection>ProQuest One Academic Eastern Edition (DO NOT USE)</collection><collection>ProQuest One Academic</collection><collection>ProQuest One Academic UKI Edition</collection><collection>ProQuest Central Basic</collection><collection>MEDLINE - Academic</collection><jtitle>Biological cybernetics</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Malekpour, Sheida</au><au>Sethares, William A.</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Conditional Granger causality and partitioned Granger causality: differences and similarities</atitle><jtitle>Biological cybernetics</jtitle><stitle>Biol Cybern</stitle><addtitle>Biol Cybern</addtitle><date>2015-12-01</date><risdate>2015</risdate><volume>109</volume><issue>6</issue><spage>627</spage><epage>637</epage><pages>627-637</pages><issn>0340-1200</issn><eissn>1432-0770</eissn><abstract>Neural information modeling and analysis often requires a measurement of the mutual influence among many signals. A common technique is the conditional Granger causality (cGC) which measures the influence of one time series on another time series in the presence of a third. Geweke has translated this condition into the frequency domain and has explored the mathematical relationships between the time and frequency domain expressions. Chen has observed that in practice, the expressions may return (meaningless) negative numbers, and has proposed an alternative which is based on a partitioned matrix scheme, which we call partitioned Granger causality (pGC). There has been some confusion in the literature about the relationship between cGC and pGC; some authors treat them as essentially identical measures, while others have noted that some properties (such as the relationship between the time and frequency domain expressions) do not hold for the pGC. This paper presents a series of matrix equalities that simplify the calculation of the pGC. In this simplified expression, the essential differences and similarities between the cGC and the pGC become clear; in essence, the pGC is dependent on only a subset of the parameters in the model estimation, and the noise residuals (which are uncorrelated in the cGC) need not be uncorrelated in the pGC. The mathematical results are illustrated with a simulation, and the measures are applied to an EEG dataset.</abstract><cop>Berlin/Heidelberg</cop><pub>Springer Berlin Heidelberg</pub><pmid>26474876</pmid><doi>10.1007/s00422-015-0665-3</doi><tpages>11</tpages></addata></record>
fulltext fulltext
identifier ISSN: 0340-1200
ispartof Biological cybernetics, 2015-12, Vol.109 (6), p.627-637
issn 0340-1200
1432-0770
language eng
recordid cdi_proquest_miscellaneous_1777997682
source MEDLINE; SpringerLink Journals - AutoHoldings
subjects Analogies
Bioinformatics
Biomedical and Life Sciences
Biomedicine
Causality
Complex Systems
Computer Appl. in Life Sciences
Computer simulation
Confusion
Cybernetics
Datasets
Electroencephalography
Frequency domains
Mathematical models
Models, Theoretical
Neurobiology
Neurology
Neurosciences
Noise
Original Article
Time series
title Conditional Granger causality and partitioned Granger causality: differences and similarities
url https://sfx.bib-bvb.de/sfx_tum?ctx_ver=Z39.88-2004&ctx_enc=info:ofi/enc:UTF-8&ctx_tim=2025-01-04T19%3A28%3A07IST&url_ver=Z39.88-2004&url_ctx_fmt=infofi/fmt:kev:mtx:ctx&rfr_id=info:sid/primo.exlibrisgroup.com:primo3-Article-proquest_cross&rft_val_fmt=info:ofi/fmt:kev:mtx:journal&rft.genre=article&rft.atitle=Conditional%20Granger%20causality%20and%20partitioned%20Granger%20causality:%20differences%20and%20similarities&rft.jtitle=Biological%20cybernetics&rft.au=Malekpour,%20Sheida&rft.date=2015-12-01&rft.volume=109&rft.issue=6&rft.spage=627&rft.epage=637&rft.pages=627-637&rft.issn=0340-1200&rft.eissn=1432-0770&rft_id=info:doi/10.1007/s00422-015-0665-3&rft_dat=%3Cproquest_cross%3E3874576991%3C/proquest_cross%3E%3Curl%3E%3C/url%3E&disable_directlink=true&sfx.directlink=off&sfx.report_link=0&rft_id=info:oai/&rft_pqid=1735596619&rft_id=info:pmid/26474876&rfr_iscdi=true