Interleukin 1-β responses to bacterial toxins and sudden infant death syndrome

We tested the hypothesis that significantly higher IL-1β responses to toxic shock syndrome toxin (TSST) noted for parents of sudden infant death syndrome (SIDS) infants might be due in part to genetic factors such as the IL-1β (C-511T) and IL-1RN (T + 2018C) single nucleotide polymorphisms (SNP). Th...

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Veröffentlicht in:	FEMS immunology and medical microbiology 2004-09, Vol.42 (1), p.139-145
Hauptverfasser:	Moscovis, Sophia M., Gordon, Ann E., Hall, Sharron T., Gleeson, Maree, Scott, Rodney J., Roberts-Thomsom, June, Weir, Donald M., Busuttil, Anthony, Blackwell, C.Caroline
Format:	Artikel
Sprache:	eng
Schlagworte:	Bacterial Toxins - immunology Cigarette smoke Continental Population Groups Endotoxins Enterotoxins - immunology Ethnic groups Frequency distribution Gene Frequency Gene polymorphisms Genetic factors Genetic Predisposition to Disease Genotype & phenotype Genotypes Humans IL-1β Incidence Infant Infants Inflammation Interleukin 1 Interleukin 1 receptors Interleukin 10 Interleukin-1 - genetics Interleukin-1 - metabolism Interleukin-1β Leukocytes Leukocytes - immunology Minority & ethnic groups Native peoples Nucleotides Polymorphism, Single Nucleotide Receptors, Interleukin-1 - genetics Septic shock SIDS Single-nucleotide polymorphism Smoking Staphylococcal enterotoxin F Sudden Infant Death - epidemiology Sudden Infant Death - genetics Sudden Infant Death - immunology Sudden infant death syndrome Superantigens - immunology Toxic shock syndrome Toxins
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