Nitric oxide in early ischaemia reperfusion injury during human orthotopic liver transplantation
Altered nitric oxide (NO) metabolism has been shown to contribute to ischemia-reperfusion (IR) injury in animal models. However, similar studies have not been performed in human liver transplantation (LT). In this study, we examined nitrate, nitrite, and nitrosothiols (NOx), NO synthases (endothelia...
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Veröffentlicht in: | Transplantation 2004-07, Vol.78 (2), p.250-256 |
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Sprache: | eng |
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