Neuropeptide Y (NPY) Potentiates Phenylephrine-Induced Mitogen-Activated Protein Kinase Activation in Primary Cardiomyocytes via NPY Y5 Receptors

Neuropeptide Y (NPY) has been shown to participate in the cardiovascualr response mediated by the sympathetic system. In this report, we investigate the growth factor properties of NPY on cardiac myocytes. Mitogen-activated protein kinases (MAPK) are key signaling molecules in the transduction of tr...

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Veröffentlicht in:	Proceedings of the National Academy of Sciences - PNAS 2000-02, Vol.97 (4), p.1595-1600
Hauptverfasser:	Pellieux, Corinne, Sauthier, Thérèse, Domenighetti, Andrea, Marsh, Donald J., Palmiter, Richard D., Hans-R. Brunner, Pedrazzini, Thierry
Format:	Artikel
Sprache:	eng
Schlagworte:	Agonists Angiotensin II - pharmacology Animals Antibodies Biological Sciences Calcium Cardiology Cardiomegaly Cells, Cultured Cellular biology Enzyme Activation - drug effects Mice Mice, Inbred C57BL Mitogen-Activated Protein Kinases - metabolism Myocardium Myocardium - enzymology Myocardium - metabolism Neuropeptide Y - pharmacology Neuropeptides Neurotransmitters Peptides Pertussis Toxin phenylephrine Phenylephrine - pharmacology Phosphorylation Protein Kinase C - metabolism Proteins Receptors Receptors, Neuropeptide Y - metabolism RNA RNA, Messenger - metabolism Tetradecanoylphorbol Acetate - pharmacology Thapsigargin - pharmacology Virulence Factors, Bordetella - pharmacology
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