Susceptibility to oral cancers with CD95 and CD95L promoter SNPs may vary with the site and gender
We investigated risk association of oral cancers (tongue and buccal mucosa cancers) with FAS (−1377G > A and FAS −670 A > G) and FASL (−844 T > C) SNPs, in males and females. A case–control study of 535 oral cancer and 525 control subjects was performed. SNPs were detected in the genomic DN...
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| creator | Daripally, Sarika Nallapalle, Sateesh Reddy Katta, Saritha Prasad, Vidudala V. T. S. |
| description | We investigated risk association of oral cancers (tongue and buccal mucosa cancers) with
FAS
(−1377G > A and
FAS
−670 A > G) and
FASL
(−844 T > C) SNPs, in males and females. A case–control study of 535 oral cancer and 525 control subjects was performed. SNPs were detected in the genomic DNA isolated from peripheral blood using PCR-RFLP. We report
FASL
−844 T > C SNPs increased risk for buccal mucosa cancer in females but not in males. On the other hand,
FAS
genotypes did not alter the risk of the cancers in both females and males. However, co-occurrence of
FAS
−1377 GA and −670 GG,
FAS
−1377 AA and −670 GG genotypes, and combined genotypes of
FAS
and
FASL
(
FAS
−1377 AA +
FAS
−670 GG +
FASL
−844 CC) alter male susceptibility towards tongue cancer. In females, combined genotypes of
FAS
(−1377GA and −670 AA) were found to be a risk factor of buccal mucosa cancer (OR = 3.27, CI = 1.28–8.36;
P
≤ 0.01).
FASL
variants (GA and AA) increased tongue cancer risk in females who were tobacco users compared to non-tobacco users. In conclusion, SNPs of the
FAS
and
FASL
might alter risk of tongue and buccal mucosa cancers differentially, in a gender-dependent manner. |
| doi_str_mv | 10.1007/s13277-015-3516-x |
| format | Article |
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FAS
(−1377G > A and
FAS
−670 A > G) and
FASL
(−844 T > C) SNPs, in males and females. A case–control study of 535 oral cancer and 525 control subjects was performed. SNPs were detected in the genomic DNA isolated from peripheral blood using PCR-RFLP. We report
FASL
−844 T > C SNPs increased risk for buccal mucosa cancer in females but not in males. On the other hand,
FAS
genotypes did not alter the risk of the cancers in both females and males. However, co-occurrence of
FAS
−1377 GA and −670 GG,
FAS
−1377 AA and −670 GG genotypes, and combined genotypes of
FAS
and
FASL
(
FAS
−1377 AA +
FAS
−670 GG +
FASL
−844 CC) alter male susceptibility towards tongue cancer. In females, combined genotypes of
FAS
(−1377GA and −670 AA) were found to be a risk factor of buccal mucosa cancer (OR = 3.27, CI = 1.28–8.36;
P
≤ 0.01).
FASL
variants (GA and AA) increased tongue cancer risk in females who were tobacco users compared to non-tobacco users. In conclusion, SNPs of the
FAS
and
FASL
might alter risk of tongue and buccal mucosa cancers differentially, in a gender-dependent manner.</description><identifier>ISSN: 1010-4283</identifier><identifier>EISSN: 1423-0380</identifier><identifier>DOI: 10.1007/s13277-015-3516-x</identifier><identifier>PMID: 25944167</identifier><language>eng</language><publisher>Dordrecht: Springer Netherlands</publisher><subject>Adolescent ; Adult ; Aged ; Aged, 80 and over ; Biomedical and Life Sciences ; Biomedicine ; Cancer Research ; Carcinoma, Squamous Cell - genetics ; Carcinoma, Squamous Cell - mortality ; Carcinoma, Squamous Cell - secondary ; Carcinoma, Verrucous - genetics ; Carcinoma, Verrucous - mortality ; Carcinoma, Verrucous - secondary ; Case-Control Studies ; Fas Ligand Protein - genetics ; fas Receptor - genetics ; Female ; Follow-Up Studies ; Gender Identity ; Genetic Predisposition to Disease ; Genotype ; Humans ; Lymphatic Metastasis ; Male ; Middle Aged ; Mouth Mucosa ; Mouth Neoplasms - genetics ; Mouth Neoplasms - mortality ; Mouth Neoplasms - pathology ; Neoplasm Invasiveness ; Neoplasm Recurrence, Local - genetics ; Neoplasm Recurrence, Local - mortality ; Neoplasm Recurrence, Local - pathology ; Neoplasm Staging ; Oral cancer ; Polymerase Chain Reaction ; Polymorphism ; Polymorphism, Restriction Fragment Length ; Polymorphism, Single Nucleotide - genetics ; Prognosis ; Promoter Regions, Genetic - genetics ; Research Article ; Risk Factors ; Survival Rate ; Tongue ; Young Adult</subject><ispartof>Tumor biology, 2015-09, Vol.36 (10), p.7817-7830</ispartof><rights>International Society of Oncology and BioMarkers (ISOBM) 2015</rights><lds50>peer_reviewed</lds50><oa>free_for_read</oa><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c442t-57627646c36fb40ea5a915bf10a87a77edcc351c872439ca99956fb25f2ca3a03</citedby><cites>FETCH-LOGICAL-c442t-57627646c36fb40ea5a915bf10a87a77edcc351c872439ca99956fb25f2ca3a03</cites></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><linktopdf>$$Uhttps://link.springer.com/content/pdf/10.1007/s13277-015-3516-x$$EPDF$$P50$$Gspringer$$H</linktopdf><linktohtml>$$Uhttps://link.springer.com/10.1007/s13277-015-3516-x$$EHTML$$P50$$Gspringer$$H</linktohtml><link.rule.ids>314,776,780,27904,27905,41468,42537,51299</link.rule.ids><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/25944167$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Daripally, Sarika</creatorcontrib><creatorcontrib>Nallapalle, Sateesh Reddy</creatorcontrib><creatorcontrib>Katta, Saritha</creatorcontrib><creatorcontrib>Prasad, Vidudala V. T. S.</creatorcontrib><title>Susceptibility to oral cancers with CD95 and CD95L promoter SNPs may vary with the site and gender</title><title>Tumor biology</title><addtitle>Tumor Biol</addtitle><addtitle>Tumour Biol</addtitle><description>We investigated risk association of oral cancers (tongue and buccal mucosa cancers) with
FAS
(−1377G > A and
FAS
−670 A > G) and
FASL
(−844 T > C) SNPs, in males and females. A case–control study of 535 oral cancer and 525 control subjects was performed. SNPs were detected in the genomic DNA isolated from peripheral blood using PCR-RFLP. We report
FASL
−844 T > C SNPs increased risk for buccal mucosa cancer in females but not in males. On the other hand,
FAS
genotypes did not alter the risk of the cancers in both females and males. However, co-occurrence of
FAS
−1377 GA and −670 GG,
FAS
−1377 AA and −670 GG genotypes, and combined genotypes of
FAS
and
FASL
(
FAS
−1377 AA +
FAS
−670 GG +
FASL
−844 CC) alter male susceptibility towards tongue cancer. In females, combined genotypes of
FAS
(−1377GA and −670 AA) were found to be a risk factor of buccal mucosa cancer (OR = 3.27, CI = 1.28–8.36;
P
≤ 0.01).
FASL
variants (GA and AA) increased tongue cancer risk in females who were tobacco users compared to non-tobacco users. In conclusion, SNPs of the
FAS
and
FASL
might alter risk of tongue and buccal mucosa cancers differentially, in a gender-dependent manner.</description><subject>Adolescent</subject><subject>Adult</subject><subject>Aged</subject><subject>Aged, 80 and over</subject><subject>Biomedical and Life Sciences</subject><subject>Biomedicine</subject><subject>Cancer Research</subject><subject>Carcinoma, Squamous Cell - genetics</subject><subject>Carcinoma, Squamous Cell - mortality</subject><subject>Carcinoma, Squamous Cell - secondary</subject><subject>Carcinoma, Verrucous - genetics</subject><subject>Carcinoma, Verrucous - mortality</subject><subject>Carcinoma, Verrucous - secondary</subject><subject>Case-Control Studies</subject><subject>Fas Ligand Protein - genetics</subject><subject>fas Receptor - genetics</subject><subject>Female</subject><subject>Follow-Up Studies</subject><subject>Gender Identity</subject><subject>Genetic Predisposition to Disease</subject><subject>Genotype</subject><subject>Humans</subject><subject>Lymphatic Metastasis</subject><subject>Male</subject><subject>Middle Aged</subject><subject>Mouth Mucosa</subject><subject>Mouth Neoplasms - genetics</subject><subject>Mouth Neoplasms - mortality</subject><subject>Mouth Neoplasms - pathology</subject><subject>Neoplasm Invasiveness</subject><subject>Neoplasm Recurrence, Local - genetics</subject><subject>Neoplasm Recurrence, Local - mortality</subject><subject>Neoplasm Recurrence, Local - pathology</subject><subject>Neoplasm Staging</subject><subject>Oral cancer</subject><subject>Polymerase Chain Reaction</subject><subject>Polymorphism</subject><subject>Polymorphism, Restriction Fragment Length</subject><subject>Polymorphism, Single Nucleotide - genetics</subject><subject>Prognosis</subject><subject>Promoter Regions, Genetic - genetics</subject><subject>Research Article</subject><subject>Risk Factors</subject><subject>Survival Rate</subject><subject>Tongue</subject><subject>Young Adult</subject><issn>1010-4283</issn><issn>1423-0380</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2015</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><sourceid>8G5</sourceid><sourceid>ABUWG</sourceid><sourceid>AFKRA</sourceid><sourceid>AZQEC</sourceid><sourceid>BENPR</sourceid><sourceid>CCPQU</sourceid><sourceid>DWQXO</sourceid><sourceid>GNUQQ</sourceid><sourceid>GUQSH</sourceid><sourceid>M2O</sourceid><recordid>eNp1kUtLAzEUhYMovn-AGwm4cTOadyZLqU8oKlTXIZNm2pF51CSj9t8bO1VEcJUL-e6599wDwBFGZxgheR4wJVJmCPOMciyyjw2wixmhGaI52kw1wihjJKc7YC-EF5RApcQ22CFcMYaF3AXFpA_WLWJVVHUVlzB2sPOmhta01vkA36s4h6NLxaFpp6tiDBe-a7roPJzcPwbYmCV8M345oHHuYKiiW-Ez106dPwBbpamDO1y_--D5-uppdJuNH27uRhfjzDJGYsalIFIwYakoC4ac4UZhXpQYmVwaKd3U2uTS5pIwqqxRSvFEEl4Sa6hBdB-cDrppv9fehaibKnmra9O6rg8aS0IUkTRXCT35g750vW_TdisKqVwxkSg8UNZ3IXhX6oWvmmRVY6S_AtBDADrdVX8FoD9Sz_FauS8aN_3p-L54AsgAhPTVzpz_Nfpf1U8CH48J</recordid><startdate>20150901</startdate><enddate>20150901</enddate><creator>Daripally, Sarika</creator><creator>Nallapalle, Sateesh Reddy</creator><creator>Katta, Saritha</creator><creator>Prasad, Vidudala V. T. S.</creator><general>Springer Netherlands</general><general>Springer Nature B.V</general><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>3V.</scope><scope>7T5</scope><scope>7TO</scope><scope>7X7</scope><scope>7XB</scope><scope>88E</scope><scope>8AO</scope><scope>8C1</scope><scope>8FE</scope><scope>8FH</scope><scope>8FI</scope><scope>8FJ</scope><scope>8FK</scope><scope>8G5</scope><scope>ABUWG</scope><scope>AFKRA</scope><scope>AZQEC</scope><scope>BBNVY</scope><scope>BENPR</scope><scope>BHPHI</scope><scope>CCPQU</scope><scope>DWQXO</scope><scope>FYUFA</scope><scope>GHDGH</scope><scope>GNUQQ</scope><scope>GUQSH</scope><scope>H94</scope><scope>HCIFZ</scope><scope>K9.</scope><scope>LK8</scope><scope>M0S</scope><scope>M1P</scope><scope>M2O</scope><scope>M7P</scope><scope>MBDVC</scope><scope>PIMPY</scope><scope>PQEST</scope><scope>PQQKQ</scope><scope>PQUKI</scope><scope>Q9U</scope><scope>7X8</scope></search><sort><creationdate>20150901</creationdate><title>Susceptibility to oral cancers with CD95 and CD95L promoter SNPs may vary with the site and gender</title><author>Daripally, Sarika ; Nallapalle, Sateesh Reddy ; Katta, Saritha ; Prasad, Vidudala V. T. S.</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c442t-57627646c36fb40ea5a915bf10a87a77edcc351c872439ca99956fb25f2ca3a03</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2015</creationdate><topic>Adolescent</topic><topic>Adult</topic><topic>Aged</topic><topic>Aged, 80 and over</topic><topic>Biomedical and Life Sciences</topic><topic>Biomedicine</topic><topic>Cancer Research</topic><topic>Carcinoma, Squamous Cell - genetics</topic><topic>Carcinoma, Squamous Cell - mortality</topic><topic>Carcinoma, Squamous Cell - secondary</topic><topic>Carcinoma, Verrucous - genetics</topic><topic>Carcinoma, Verrucous - mortality</topic><topic>Carcinoma, Verrucous - secondary</topic><topic>Case-Control Studies</topic><topic>Fas Ligand Protein - genetics</topic><topic>fas Receptor - genetics</topic><topic>Female</topic><topic>Follow-Up Studies</topic><topic>Gender Identity</topic><topic>Genetic Predisposition to Disease</topic><topic>Genotype</topic><topic>Humans</topic><topic>Lymphatic Metastasis</topic><topic>Male</topic><topic>Middle Aged</topic><topic>Mouth Mucosa</topic><topic>Mouth Neoplasms - genetics</topic><topic>Mouth Neoplasms - mortality</topic><topic>Mouth Neoplasms - pathology</topic><topic>Neoplasm Invasiveness</topic><topic>Neoplasm Recurrence, Local - genetics</topic><topic>Neoplasm Recurrence, Local - mortality</topic><topic>Neoplasm Recurrence, Local - pathology</topic><topic>Neoplasm Staging</topic><topic>Oral cancer</topic><topic>Polymerase Chain Reaction</topic><topic>Polymorphism</topic><topic>Polymorphism, Restriction Fragment Length</topic><topic>Polymorphism, Single Nucleotide - genetics</topic><topic>Prognosis</topic><topic>Promoter Regions, Genetic - genetics</topic><topic>Research Article</topic><topic>Risk Factors</topic><topic>Survival Rate</topic><topic>Tongue</topic><topic>Young Adult</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Daripally, Sarika</creatorcontrib><creatorcontrib>Nallapalle, Sateesh Reddy</creatorcontrib><creatorcontrib>Katta, Saritha</creatorcontrib><creatorcontrib>Prasad, Vidudala V. 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T. S.</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Susceptibility to oral cancers with CD95 and CD95L promoter SNPs may vary with the site and gender</atitle><jtitle>Tumor biology</jtitle><stitle>Tumor Biol</stitle><addtitle>Tumour Biol</addtitle><date>2015-09-01</date><risdate>2015</risdate><volume>36</volume><issue>10</issue><spage>7817</spage><epage>7830</epage><pages>7817-7830</pages><issn>1010-4283</issn><eissn>1423-0380</eissn><abstract>We investigated risk association of oral cancers (tongue and buccal mucosa cancers) with
FAS
(−1377G > A and
FAS
−670 A > G) and
FASL
(−844 T > C) SNPs, in males and females. A case–control study of 535 oral cancer and 525 control subjects was performed. SNPs were detected in the genomic DNA isolated from peripheral blood using PCR-RFLP. We report
FASL
−844 T > C SNPs increased risk for buccal mucosa cancer in females but not in males. On the other hand,
FAS
genotypes did not alter the risk of the cancers in both females and males. However, co-occurrence of
FAS
−1377 GA and −670 GG,
FAS
−1377 AA and −670 GG genotypes, and combined genotypes of
FAS
and
FASL
(
FAS
−1377 AA +
FAS
−670 GG +
FASL
−844 CC) alter male susceptibility towards tongue cancer. In females, combined genotypes of
FAS
(−1377GA and −670 AA) were found to be a risk factor of buccal mucosa cancer (OR = 3.27, CI = 1.28–8.36;
P
≤ 0.01).
FASL
variants (GA and AA) increased tongue cancer risk in females who were tobacco users compared to non-tobacco users. In conclusion, SNPs of the
FAS
and
FASL
might alter risk of tongue and buccal mucosa cancers differentially, in a gender-dependent manner.</abstract><cop>Dordrecht</cop><pub>Springer Netherlands</pub><pmid>25944167</pmid><doi>10.1007/s13277-015-3516-x</doi><tpages>14</tpages><oa>free_for_read</oa></addata></record> |
| fulltext | fulltext |
| identifier | ISSN: 1010-4283 |
| ispartof | Tumor biology, 2015-09, Vol.36 (10), p.7817-7830 |
| issn | 1010-4283 1423-0380 |
| language | eng |
| recordid | cdi_proquest_miscellaneous_1722927389 |
| source | MEDLINE; Springer Nature - Complete Springer Journals |
| subjects | Adolescent Adult Aged Aged, 80 and over Biomedical and Life Sciences Biomedicine Cancer Research Carcinoma, Squamous Cell - genetics Carcinoma, Squamous Cell - mortality Carcinoma, Squamous Cell - secondary Carcinoma, Verrucous - genetics Carcinoma, Verrucous - mortality Carcinoma, Verrucous - secondary Case-Control Studies Fas Ligand Protein - genetics fas Receptor - genetics Female Follow-Up Studies Gender Identity Genetic Predisposition to Disease Genotype Humans Lymphatic Metastasis Male Middle Aged Mouth Mucosa Mouth Neoplasms - genetics Mouth Neoplasms - mortality Mouth Neoplasms - pathology Neoplasm Invasiveness Neoplasm Recurrence, Local - genetics Neoplasm Recurrence, Local - mortality Neoplasm Recurrence, Local - pathology Neoplasm Staging Oral cancer Polymerase Chain Reaction Polymorphism Polymorphism, Restriction Fragment Length Polymorphism, Single Nucleotide - genetics Prognosis Promoter Regions, Genetic - genetics Research Article Risk Factors Survival Rate Tongue Young Adult |
| title | Susceptibility to oral cancers with CD95 and CD95L promoter SNPs may vary with the site and gender |
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