Suppression of the TRIF-dependent signaling pathway of Toll-like receptor by CDr10b in RAW264.7 macrophages

Toll-like receptors (TLRs) recognize distinct pathogen-associated molecular patterns and play a critical role in innate immune responses. TLR signaling pathways can be largely classified as either myeloid differential factor 88 (MyD88)- or toll-interleukin-1 receptor domain-containing adapter induci...

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Veröffentlicht in:	International immunopharmacology 2015-09, Vol.28 (1), p.29-33
Hauptverfasser:	Gu, Gyo-Jeong, Ahn, Sang-il, Kim, Ji-Soo, Hong, Chae-Yeon, Lee, Sung-Chan, Chang, Young-Tae, Choi, Tae Hyun, Kim, Byoung Soo, Youn, Hyung-Sun
Format:	Artikel
Sprache:	eng
Schlagworte:	Adaptor Proteins, Vesicular Transport - drug effects Animals Boron Compounds - chemical synthesis Boron Compounds - pharmacology CDr10b Chemokine CXCL10 - biosynthesis Cyclooxygenase 2 - drug effects Interferon Regulatory Factor-3 - biosynthesis Interferon Regulatory Factor-3 - genetics IP-10 IRF3 Macrophages - drug effects Mice NF-kappa B - drug effects Nitric Oxide Synthase Type II - drug effects Protein-Serine-Threonine Kinases - biosynthesis Protein-Serine-Threonine Kinases - genetics RAW 264.7 Cells Signal Transduction - drug effects Toll-like receptor Toll-Like Receptor 3 - agonists Toll-Like Receptor 4 - agonists Toll-Like Receptors - antagonists & inhibitors TRIF
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