Differential response of normal and HPV immortalized ectocervical epithelial cells to B[a]P

Cigarette smoking has been established as a risk factor for the development of cervical cancer. Polycydlic aromatic hydrocarbons such as benzo (B[a]P) which are present in cigarette smoke, might account for this increased risk. The effects of B[a]P on cell growth, aryl hydrocarbon hydroxylase, DNA a...

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Veröffentlicht in:	Carcinogenesis (New York) 1995-10, Vol.16 (10), p.2413-2418
Hauptverfasser:	Sizemore, Nywana, Mukhtar, Hasan, Couch, Letha H., Howard, Paul C., Rorke, Ellen A.
Format:	Artikel
Sprache:	eng
Schlagworte:	Benzo(a)pyrene - metabolism Benzo(a)pyrene - toxicity Biological and medical sciences Carcinogenesis, carcinogens and anticarcinogens Carcinogens - metabolism Carcinogens - toxicity Cell Division - drug effects Cell Line, Transformed Cell Transformation, Viral Cervix Uteri - cytology Cervix Uteri - drug effects Cervix Uteri - pathology Chemical agents DNA Adducts - metabolism Epithelial Cells Epithelium - drug effects Epithelium - pathology Female Humans Kinetics Medical sciences Papillomaviridae - genetics Precancerous Conditions - chemically induced Precancerous Conditions - pathology Risk Factors Smoking - adverse effects Time Factors Tumor Suppressor Protein p53 - metabolism Tumors Uterine Cervical Neoplasms - chemically induced Uterine Cervical Neoplasms - epidemiology Uterine Cervical Neoplasms - pathology
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