P2Y2 nucleotide receptor-mediated extracellular signal-regulated kinases and protein kinase C activation induces the invasion of highly metastatic breast cancer cells

Tumor metastasis is considered the main cause of mortality in cancer patients, thus it is important to investigate the differences between high- and low-metastatic cancer cells. Our previous study showed that the highly metastatic breast cancer cell line MDA-MB-231 released higher levels of ATP and...

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Veröffentlicht in:	Oncology reports 2015-07, Vol.34 (1), p.195-202
Hauptverfasser:	EUN, SO YOUNG, KO, YOUNG SHIN, PARK, SANG WON, CHANG, KI CHURL, KIM, HYE JUNG
Format:	Artikel
Sprache:	eng
Schlagworte:	adenosine triphosphate Breast cancer Breast Neoplasms - genetics Breast Neoplasms - pathology Cell adhesion & migration Cell culture Cell Proliferation - genetics epithelial-mesenchymal transition Epithelial-Mesenchymal Transition - genetics extracellular signal-regulated kinase Female Gene Expression Regulation, Neoplastic Growth factors Humans Immunoglobulins invasion Kinases MCF-7 Cells Metastasis Neoplasm Metastasis P2Y2 receptor Penicillin Phosphorylation protein kinase C Protein Kinase C - biosynthesis Protein Kinase C - genetics Protein-Lysine 6-Oxidase - biosynthesis Proteins Receptors, Purinergic P2Y2 - biosynthesis Receptors, Purinergic P2Y2 - genetics Signal Transduction Studies Tumor Microenvironment - genetics
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