Poststreptococcal Glomerulonephritis in a Patient with Essential Thrombocytosis
In addition to being the main cause of glomerulonephritis in children, poststreptococcal glomerulonephritis (PSGN) has recently been shown in older patients, especially those with malignancy or diabetes mellitus. The pathogenesis of PSGN has been ascribed to activation of complement 3 (C3) of the al...
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| Veröffentlicht in: | Journal of Nippon Medical School 2015/02/15, Vol.82(1), pp.59-63 |
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| container_title | Journal of Nippon Medical School |
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| creator | Ulusoy, Sukru Ozkan, Gulsum Sonmez, Mehmet Mungan, Sevdegül Köseoğlu, Rahman Cansız, Muammer Kaynar, Kübra |
| description | In addition to being the main cause of glomerulonephritis in children, poststreptococcal glomerulonephritis (PSGN) has recently been shown in older patients, especially those with malignancy or diabetes mellitus. The pathogenesis of PSGN has been ascribed to activation of complement 3 (C3) of the alternative complement cascade which, along with immunoglobulin (Ig) G and IgM deposits, is observed in renal tissue. Our aim here is to discuss the probable causes of PSGN developing with isolated IgM deposition in a 52-year-old patient with essential thrombocytosis followed-up over the previous 3.5 years. These characteristics make our case the first to be reported in the literature. |
| doi_str_mv | 10.1272/jnms.82.59 |
| format | Article |
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The pathogenesis of PSGN has been ascribed to activation of complement 3 (C3) of the alternative complement cascade which, along with immunoglobulin (Ig) G and IgM deposits, is observed in renal tissue. Our aim here is to discuss the probable causes of PSGN developing with isolated IgM deposition in a 52-year-old patient with essential thrombocytosis followed-up over the previous 3.5 years. 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The pathogenesis of PSGN has been ascribed to activation of complement 3 (C3) of the alternative complement cascade which, along with immunoglobulin (Ig) G and IgM deposits, is observed in renal tissue. Our aim here is to discuss the probable causes of PSGN developing with isolated IgM deposition in a 52-year-old patient with essential thrombocytosis followed-up over the previous 3.5 years. 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The pathogenesis of PSGN has been ascribed to activation of complement 3 (C3) of the alternative complement cascade which, along with immunoglobulin (Ig) G and IgM deposits, is observed in renal tissue. Our aim here is to discuss the probable causes of PSGN developing with isolated IgM deposition in a 52-year-old patient with essential thrombocytosis followed-up over the previous 3.5 years. These characteristics make our case the first to be reported in the literature.</abstract><cop>Japan</cop><pub>The Medical Association of Nippon Medical School</pub><pmid>25797878</pmid><doi>10.1272/jnms.82.59</doi><tpages>5</tpages><oa>free_for_read</oa></addata></record> |
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| subjects | Biomarkers - analysis Biopsy Complement C3 - analysis essential thrombocytosis Fluorescent Antibody Technique glomerulonephritis Glomerulonephritis - diagnosis Glomerulonephritis - drug therapy Glomerulonephritis - etiology Glomerulonephritis - immunology Glucocorticoids - therapeutic use Hematologic Agents - therapeutic use Humans Immunoglobulin M - analysis Kidney - drug effects Kidney - immunology Kidney - ultrastructure Male Microscopy, Electron Middle Aged myeloproliferative neoplasm poststreptococcal glomerulonephritis Predictive Value of Tests Risk Factors Streptococcal Infections - complications Thrombocythemia, Essential - diagnosis Thrombocythemia, Essential - drug therapy Thrombocythemia, Essential - etiology Thrombocythemia, Essential - immunology Time Factors Treatment Outcome |
| title | Poststreptococcal Glomerulonephritis in a Patient with Essential Thrombocytosis |
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