Stress response protein cirp links inflammation and tumorigenesis in colitis-associated cancer

Colitis-associated cancer (CAC) is caused by chronic intestinal inflammation and is reported to be associated with refractory inflammatory bowel disease (IBD). Defective apoptosis of inflammatory cell populations seems to be a relevant pathogenetic mechanism in refractory IBD. We assessed the involv...

Ausführliche Beschreibung

Gespeichert in:

Bibliographische Detailangaben
Veröffentlicht in:	Cancer research (Chicago, Ill.) Ill.), 2014-11, Vol.74 (21), p.6119-6128
Hauptverfasser:	Sakurai, Toshiharu, Kashida, Hiroshi, Watanabe, Tomohiro, Hagiwara, Satoru, Mizushima, Tsunekazu, Iijima, Hideki, Nishida, Naoshi, Higashitsuji, Hiroaki, Fujita, Jun, Kudo, Masatoshi
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Apoptosis - genetics Carcinogenesis - genetics Colitis - complications Colitis - genetics Colitis - pathology Colorectal Neoplasms - etiology Colorectal Neoplasms - genetics Colorectal Neoplasms - pathology Disease Models, Animal Gene Expression Regulation, Neoplastic Humans Inflammatory Bowel Diseases - complications Inflammatory Bowel Diseases - genetics Inflammatory Bowel Diseases - pathology Interleukin-23 Subunit p19 Mice Neoplasm Proteins - biosynthesis RNA-Binding Proteins - biosynthesis RNA-Binding Proteins - genetics Tumor Necrosis Factor-alpha - genetics Tumor Necrosis Factor-alpha - metabolism
Online-Zugang:	Volltext
Tags:	Tag hinzufügen Keine Tags, Fügen Sie den ersten Tag hinzu!

Schreiben Sie den ersten Kommentar!