Inhibition of vascular endothelial growth factor-induced endothelial cell migration by ETS1 antisense oligonucleotides

Vascular endothelial growth factor (VEGF) increased the level of ETS1 mRNA in human umbilical vein endothelial cells (HUVEC) and human lung microvascular endothelial cells (HMVEC-L) over 5-fold. Protein levels were shown to increase concordantly. VEGF was also found to stimulate the invasiveness of...

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Veröffentlicht in:	Cancer research (Chicago, Ill.) Ill.), 1997-05, Vol.57 (10), p.2013-2019
Hauptverfasser:	CHEN, Z.-Q, FISHER, R. J, RIGGS, C. W, RHIM, J. S, LAUTENBERGER, J. A
Format:	Artikel
Sprache:	eng
Schlagworte:	Biological and medical sciences Cell Division - drug effects Cell Movement - drug effects Cell physiology Cell transformation and carcinogenesis. Action of oncogenes and antioncogenes Cells, Cultured Collagen Drug Combinations Endothelial Growth Factors - antagonists & inhibitors Endothelial Growth Factors - pharmacology Endothelium, Vascular - cytology Endothelium, Vascular - drug effects Fundamental and applied biological sciences. Psychology Gelatin Gene Expression - drug effects Gene Expression Regulation - drug effects Humans Laminin Lymphokines - antagonists & inhibitors Lymphokines - pharmacology Membranes Molecular and cellular biology Oligonucleotides, Antisense - pharmacology Proteoglycans Proto-Oncogene Protein c-ets-1 Proto-Oncogene Proteins - biosynthesis Proto-Oncogene Proteins - genetics Proto-Oncogene Proteins c-ets RNA, Messenger - biosynthesis RNA, Messenger - genetics RNA, Messenger - metabolism Stimulation, Chemical Tetradecanoylphorbol Acetate - pharmacology Transcription Factors - biosynthesis Transcription Factors - genetics Urokinase-Type Plasminogen Activator - drug effects Vascular Endothelial Growth Factor A Vascular Endothelial Growth Factors
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