Endotoxin Pretreatment Increases Endogenous Myocardial Catalase Activity and Decreases Ischemia-Reperfusion Injury of Isolated Rat Hearts

Hearts isolated from rats pretreated 24 hr before with endotoxin had increased myocardial catalase activity, but the same superoxide dismutase, glutathione peroxidase, glutathione reductase, and glucose-6-phosphate dehydrogenase activities, as hearts from untreated rats. Hearts isolated from rats pr...

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Veröffentlicht in:	Proceedings of the National Academy of Sciences - PNAS 1989-04, Vol.86 (7), p.2516-2520
Hauptverfasser:	Brown, James M., Grosso, Michael A., Terada, Lance S., Glenn J. R. Whitman, Banerjee, Anirban, White, Carl W., Harken, Alden H., Repine, John E.
Format:	Artikel
Sprache:	eng
Schlagworte:	Amitrole - pharmacology Animals Biological and medical sciences Blood Pressure - drug effects Catalase - metabolism Cell physiology Endotoxins Endotoxins - pharmacology Fundamental and applied biological sciences. Psychology Heart Heart - drug effects Heart - physiology Heart - physiopathology Heart Ventricles - physiopathology In Vitro Techniques Ischemia Lungs Male Molecular and cellular biology Myocardial reperfusion Myocardial Reperfusion Injury - enzymology Myocardial Reperfusion Injury - prevention & control Myocardium - enzymology Perfusion Pretreatment Rats Rats, Inbred Strains Reperfusion injury Responses to growth factors, tumor promotors, other factors Salmonella typhimurium Superoxides Triazoles - pharmacology Ventricular function
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