Redox modulation of NMDA receptor-mediated toxicity in mammalian central neurons

Acute neurological injury from hypoxia-ischemia, hypoglycemia, and trauma is thought to be predominantly mediated by activation of the N- methyl- d-aspartate (NMDA) subtype of glutamate receptor in the brain and the subsequent influx of calcium ions through receptor-operated channels. Several chroni...

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Veröffentlicht in:	Neuroscience letters 1990-03, Vol.110 (3), p.291-296
Hauptverfasser:	Levy, Daniel I., Sucher, Nikolaus J., Lipton, Stuart A.
Format:	Artikel
Sprache:	eng
Schlagworte:	2-Amino-5-phosphonovalerate - pharmacology Action Potentials - drug effects Animals Biological and medical sciences Cell culture Cells, Cultured Chemical oxidation Chemical reduction Dithionitrobenzoic Acid - pharmacology Dithiothreitol - toxicity Excitotoxicity Fundamental and applied biological sciences. Psychology Isolated neuron and nerve. Neuroglia Oxidation-Reduction Patch clamp Postnatal rat retinal ganglion cell Rats Receptors, N-Methyl-D-Aspartate Receptors, Neurotransmitter - drug effects Receptors, Neurotransmitter - physiology Retina - physiology Retinal Ganglion Cells - cytology Retinal Ganglion Cells - drug effects Retinal Ganglion Cells - physiology Vertebrates: nervous system and sense organs
Online-Zugang:	Volltext
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