Tumor necrosis factor-related apoptosis-inducing ligand mediates the resolution of allergic airway inflammation induced by chronic allergen inhalation

Allergic asthma can vanish over time either spontaneously or induced by allergen-specific immunotherapy. In mice with established airway allergic inflammation, chronic intranasal (IN) allergen challenges decreases progressively airway allergic inflammation. Here we compared the contribution of diffe...

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Veröffentlicht in:	Mucosal immunology 2014-09, Vol.7 (5), p.1199-1208
Hauptverfasser:	Faustino, L, Fonseca, D M, Florsheim, E B, Resende, R R, Lepique, A P, Faquim-Mauro, E, Gomes, E, Silva, J S, Yagita, H, Russo, M
Format:	Artikel
Sprache:	eng
Schlagworte:	631/250/249/2510/31 631/250/249/2510/9 631/250/347 631/80/86 Allergens - immunology Allergology Animals Antibodies Antibodies, Monoclonal - immunology Apoptosis - immunology Biomedical and Life Sciences Biomedicine Caspase 3 - genetics Caspase 3 - metabolism Caspase 8 - genetics Caspase 8 - metabolism Chronic Disease Female Gastroenterology Gene Expression Regulation - immunology Immunology Inflammation - drug therapy Inflammation - immunology Lung - immunology Lung - physiopathology Mice Mice, Knockout Recombinant Proteins - genetics Respiratory Hypersensitivity - drug therapy Respiratory Hypersensitivity - immunology Th2 Cells - immunology TNF-Related Apoptosis-Inducing Ligand - genetics TNF-Related Apoptosis-Inducing Ligand - metabolism TNF-Related Apoptosis-Inducing Ligand - therapeutic use
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