Classification, diagnosis, and approach to treatment for angioedema: consensus report from the Hereditary Angioedema International Working Group
Angioedema is defined as localized and self‐limiting edema of the subcutaneous and submucosal tissue, due to a temporary increase in vascular permeability caused by the release of vasoactive mediator(s). When angioedema recurs without significant wheals, the patient should be diagnosed to have angio...
Gespeichert in:
| Veröffentlicht in: | Allergy (Copenhagen) 2014-05, Vol.69 (5), p.602-616 |
|---|---|
| Hauptverfasser: | , , , , , , , , , , , , , |
| Format: | Artikel |
| Sprache: | eng |
| Schlagworte: | |
| Online-Zugang: | Volltext |
| Tags: |
Tag hinzufügen
Keine Tags, Fügen Sie den ersten Tag hinzu!
|
| container_end_page | 616 |
|---|---|
| container_issue | 5 |
| container_start_page | 602 |
| container_title | Allergy (Copenhagen) |
| container_volume | 69 |
| creator | Cicardi, M. Aberer, W. Banerji, A. Bas, M. Bernstein, J. A. Bork, K. Caballero, T. Farkas, H. Grumach, A. Kaplan, A. P. Riedl, M. A. Triggiani, M. Zanichelli, A. Zuraw, B. |
| description | Angioedema is defined as localized and self‐limiting edema of the subcutaneous and submucosal tissue, due to a temporary increase in vascular permeability caused by the release of vasoactive mediator(s). When angioedema recurs without significant wheals, the patient should be diagnosed to have angioedema as a distinct disease. In the absence of accepted classification, different types of angioedema are not uniquely identified. For this reason, the European Academy of Allergy and Clinical Immunology gave its patronage to a consensus conference aimed at classifying angioedema. Four types of acquired and three types of hereditary angioedema were identified as separate forms from the analysis of the literature and were presented in detail at the meeting. Here, we summarize the analysis of the data and the resulting classification of angioedema. |
| doi_str_mv | 10.1111/all.12380 |
| format | Article |
| fullrecord | <record><control><sourceid>proquest_pubme</sourceid><recordid>TN_cdi_proquest_miscellaneous_1520362408</recordid><sourceformat>XML</sourceformat><sourcesystem>PC</sourcesystem><sourcerecordid>3268309841</sourcerecordid><originalsourceid>FETCH-LOGICAL-j4130-8dfe1b993853ab08bba98bdd9faaca1dab3551850ae5809e0b71bdf4d984cfc53</originalsourceid><addsrcrecordid>eNqNkU1vFDEMhiNERZfCgT-AInHh0GnzuZtwW636Ja3USyuOI8_Es80ykwzJjFD_BT-ZdFt64IQvtvQ-tmW_hHzi7IyXOIe-P-NCGvaGLLi0prLW6rdkwTjTldLSHJP3Oe8ZYyth2TtyLNRyJdVSL8jvTQ85-863MPkYTqnzsAsx-3xKITgK45gitA90inRKCNOAYaJdTEXd-YgOB_hG2xgyhjxnmnCMqQApDnR6QHqNCZ2fID3S9WsDvQkTpnDYCD39HtMPH3b0KsV5_ECOOugzfnzJJ-T-8uJuc11tb69uNutttVdcssq4DnljrTRaQsNM04A1jXO2A2iBO2ik1txoBqgNs8iaFW9cp5w1qu1aLU_I1-e55b6fM-apHnxuse8hYJxzzbVgcikUM_-BcqWEluJp6pd_0H2cy6X9gZLCCL1aFurzCzU3A7p6TH4oH6r_2lKA82fgl-_x8VXnrH7yuy5-1we_6_V2eyjkHw0Un08</addsrcrecordid><sourcetype>Aggregation Database</sourcetype><iscdi>true</iscdi><recordtype>article</recordtype><pqid>1513282576</pqid></control><display><type>article</type><title>Classification, diagnosis, and approach to treatment for angioedema: consensus report from the Hereditary Angioedema International Working Group</title><source>Wiley Free Content</source><source>MEDLINE</source><source>Wiley Online Library Journals Frontfile Complete</source><source>Elektronische Zeitschriftenbibliothek - Frei zugängliche E-Journals</source><creator>Cicardi, M. ; Aberer, W. ; Banerji, A. ; Bas, M. ; Bernstein, J. A. ; Bork, K. ; Caballero, T. ; Farkas, H. ; Grumach, A. ; Kaplan, A. P. ; Riedl, M. A. ; Triggiani, M. ; Zanichelli, A. ; Zuraw, B.</creator><creatorcontrib>Cicardi, M. ; Aberer, W. ; Banerji, A. ; Bas, M. ; Bernstein, J. A. ; Bork, K. ; Caballero, T. ; Farkas, H. ; Grumach, A. ; Kaplan, A. P. ; Riedl, M. A. ; Triggiani, M. ; Zanichelli, A. ; Zuraw, B. ; HAWK under the patronage of EAACI (European Academy of Allergy and Clinical Immunology)</creatorcontrib><description>Angioedema is defined as localized and self‐limiting edema of the subcutaneous and submucosal tissue, due to a temporary increase in vascular permeability caused by the release of vasoactive mediator(s). When angioedema recurs without significant wheals, the patient should be diagnosed to have angioedema as a distinct disease. In the absence of accepted classification, different types of angioedema are not uniquely identified. For this reason, the European Academy of Allergy and Clinical Immunology gave its patronage to a consensus conference aimed at classifying angioedema. Four types of acquired and three types of hereditary angioedema were identified as separate forms from the analysis of the literature and were presented in detail at the meeting. Here, we summarize the analysis of the data and the resulting classification of angioedema.</description><identifier>ISSN: 0105-4538</identifier><identifier>EISSN: 1398-9995</identifier><identifier>DOI: 10.1111/all.12380</identifier><identifier>PMID: 24673465</identifier><language>eng</language><publisher>Denmark: Blackwell Publishing Ltd</publisher><subject>Allergies ; angioedema ; Angioedema - diagnosis ; Angioedema - drug therapy ; Angioedema - etiology ; Classification ; clinical immunology ; dermatology ; education ; Humans ; urticaria</subject><ispartof>Allergy (Copenhagen), 2014-05, Vol.69 (5), p.602-616</ispartof><rights>2014 John Wiley & Sons A/S. Published by John Wiley & Sons Ltd</rights><rights>2014 John Wiley & Sons A/S. Published by John Wiley & Sons Ltd.</rights><rights>Copyright © 2014 John Wiley & Sons A/S. Published by John Wiley & Sons Ltd</rights><lds50>peer_reviewed</lds50><oa>free_for_read</oa><woscitedreferencessubscribed>false</woscitedreferencessubscribed></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><linktopdf>$$Uhttps://onlinelibrary.wiley.com/doi/pdf/10.1111%2Fall.12380$$EPDF$$P50$$Gwiley$$H</linktopdf><linktohtml>$$Uhttps://onlinelibrary.wiley.com/doi/full/10.1111%2Fall.12380$$EHTML$$P50$$Gwiley$$H</linktohtml><link.rule.ids>309,310,314,776,780,785,786,1411,1427,23909,23910,25118,27901,27902,45550,45551,46384,46808</link.rule.ids><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/24673465$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Cicardi, M.</creatorcontrib><creatorcontrib>Aberer, W.</creatorcontrib><creatorcontrib>Banerji, A.</creatorcontrib><creatorcontrib>Bas, M.</creatorcontrib><creatorcontrib>Bernstein, J. A.</creatorcontrib><creatorcontrib>Bork, K.</creatorcontrib><creatorcontrib>Caballero, T.</creatorcontrib><creatorcontrib>Farkas, H.</creatorcontrib><creatorcontrib>Grumach, A.</creatorcontrib><creatorcontrib>Kaplan, A. P.</creatorcontrib><creatorcontrib>Riedl, M. A.</creatorcontrib><creatorcontrib>Triggiani, M.</creatorcontrib><creatorcontrib>Zanichelli, A.</creatorcontrib><creatorcontrib>Zuraw, B.</creatorcontrib><creatorcontrib>HAWK under the patronage of EAACI (European Academy of Allergy and Clinical Immunology)</creatorcontrib><title>Classification, diagnosis, and approach to treatment for angioedema: consensus report from the Hereditary Angioedema International Working Group</title><title>Allergy (Copenhagen)</title><addtitle>Allergy</addtitle><description>Angioedema is defined as localized and self‐limiting edema of the subcutaneous and submucosal tissue, due to a temporary increase in vascular permeability caused by the release of vasoactive mediator(s). When angioedema recurs without significant wheals, the patient should be diagnosed to have angioedema as a distinct disease. In the absence of accepted classification, different types of angioedema are not uniquely identified. For this reason, the European Academy of Allergy and Clinical Immunology gave its patronage to a consensus conference aimed at classifying angioedema. Four types of acquired and three types of hereditary angioedema were identified as separate forms from the analysis of the literature and were presented in detail at the meeting. Here, we summarize the analysis of the data and the resulting classification of angioedema.</description><subject>Allergies</subject><subject>angioedema</subject><subject>Angioedema - diagnosis</subject><subject>Angioedema - drug therapy</subject><subject>Angioedema - etiology</subject><subject>Classification</subject><subject>clinical immunology</subject><subject>dermatology</subject><subject>education</subject><subject>Humans</subject><subject>urticaria</subject><issn>0105-4538</issn><issn>1398-9995</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2014</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNqNkU1vFDEMhiNERZfCgT-AInHh0GnzuZtwW636Ja3USyuOI8_Es80ykwzJjFD_BT-ZdFt64IQvtvQ-tmW_hHzi7IyXOIe-P-NCGvaGLLi0prLW6rdkwTjTldLSHJP3Oe8ZYyth2TtyLNRyJdVSL8jvTQ85-863MPkYTqnzsAsx-3xKITgK45gitA90inRKCNOAYaJdTEXd-YgOB_hG2xgyhjxnmnCMqQApDnR6QHqNCZ2fID3S9WsDvQkTpnDYCD39HtMPH3b0KsV5_ECOOugzfnzJJ-T-8uJuc11tb69uNutttVdcssq4DnljrTRaQsNM04A1jXO2A2iBO2ik1txoBqgNs8iaFW9cp5w1qu1aLU_I1-e55b6fM-apHnxuse8hYJxzzbVgcikUM_-BcqWEluJp6pd_0H2cy6X9gZLCCL1aFurzCzU3A7p6TH4oH6r_2lKA82fgl-_x8VXnrH7yuy5-1we_6_V2eyjkHw0Un08</recordid><startdate>201405</startdate><enddate>201405</enddate><creator>Cicardi, M.</creator><creator>Aberer, W.</creator><creator>Banerji, A.</creator><creator>Bas, M.</creator><creator>Bernstein, J. A.</creator><creator>Bork, K.</creator><creator>Caballero, T.</creator><creator>Farkas, H.</creator><creator>Grumach, A.</creator><creator>Kaplan, A. P.</creator><creator>Riedl, M. A.</creator><creator>Triggiani, M.</creator><creator>Zanichelli, A.</creator><creator>Zuraw, B.</creator><general>Blackwell Publishing Ltd</general><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>7T5</scope><scope>H94</scope><scope>K9.</scope><scope>7X8</scope></search><sort><creationdate>201405</creationdate><title>Classification, diagnosis, and approach to treatment for angioedema: consensus report from the Hereditary Angioedema International Working Group</title><author>Cicardi, M. ; Aberer, W. ; Banerji, A. ; Bas, M. ; Bernstein, J. A. ; Bork, K. ; Caballero, T. ; Farkas, H. ; Grumach, A. ; Kaplan, A. P. ; Riedl, M. A. ; Triggiani, M. ; Zanichelli, A. ; Zuraw, B.</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-j4130-8dfe1b993853ab08bba98bdd9faaca1dab3551850ae5809e0b71bdf4d984cfc53</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2014</creationdate><topic>Allergies</topic><topic>angioedema</topic><topic>Angioedema - diagnosis</topic><topic>Angioedema - drug therapy</topic><topic>Angioedema - etiology</topic><topic>Classification</topic><topic>clinical immunology</topic><topic>dermatology</topic><topic>education</topic><topic>Humans</topic><topic>urticaria</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Cicardi, M.</creatorcontrib><creatorcontrib>Aberer, W.</creatorcontrib><creatorcontrib>Banerji, A.</creatorcontrib><creatorcontrib>Bas, M.</creatorcontrib><creatorcontrib>Bernstein, J. A.</creatorcontrib><creatorcontrib>Bork, K.</creatorcontrib><creatorcontrib>Caballero, T.</creatorcontrib><creatorcontrib>Farkas, H.</creatorcontrib><creatorcontrib>Grumach, A.</creatorcontrib><creatorcontrib>Kaplan, A. P.</creatorcontrib><creatorcontrib>Riedl, M. A.</creatorcontrib><creatorcontrib>Triggiani, M.</creatorcontrib><creatorcontrib>Zanichelli, A.</creatorcontrib><creatorcontrib>Zuraw, B.</creatorcontrib><creatorcontrib>HAWK under the patronage of EAACI (European Academy of Allergy and Clinical Immunology)</creatorcontrib><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>Immunology Abstracts</collection><collection>AIDS and Cancer Research Abstracts</collection><collection>ProQuest Health & Medical Complete (Alumni)</collection><collection>MEDLINE - Academic</collection><jtitle>Allergy (Copenhagen)</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Cicardi, M.</au><au>Aberer, W.</au><au>Banerji, A.</au><au>Bas, M.</au><au>Bernstein, J. A.</au><au>Bork, K.</au><au>Caballero, T.</au><au>Farkas, H.</au><au>Grumach, A.</au><au>Kaplan, A. P.</au><au>Riedl, M. A.</au><au>Triggiani, M.</au><au>Zanichelli, A.</au><au>Zuraw, B.</au><aucorp>HAWK under the patronage of EAACI (European Academy of Allergy and Clinical Immunology)</aucorp><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Classification, diagnosis, and approach to treatment for angioedema: consensus report from the Hereditary Angioedema International Working Group</atitle><jtitle>Allergy (Copenhagen)</jtitle><addtitle>Allergy</addtitle><date>2014-05</date><risdate>2014</risdate><volume>69</volume><issue>5</issue><spage>602</spage><epage>616</epage><pages>602-616</pages><issn>0105-4538</issn><eissn>1398-9995</eissn><abstract>Angioedema is defined as localized and self‐limiting edema of the subcutaneous and submucosal tissue, due to a temporary increase in vascular permeability caused by the release of vasoactive mediator(s). When angioedema recurs without significant wheals, the patient should be diagnosed to have angioedema as a distinct disease. In the absence of accepted classification, different types of angioedema are not uniquely identified. For this reason, the European Academy of Allergy and Clinical Immunology gave its patronage to a consensus conference aimed at classifying angioedema. Four types of acquired and three types of hereditary angioedema were identified as separate forms from the analysis of the literature and were presented in detail at the meeting. Here, we summarize the analysis of the data and the resulting classification of angioedema.</abstract><cop>Denmark</cop><pub>Blackwell Publishing Ltd</pub><pmid>24673465</pmid><doi>10.1111/all.12380</doi><tpages>15</tpages><oa>free_for_read</oa></addata></record> |
| fulltext | fulltext |
| identifier | ISSN: 0105-4538 |
| ispartof | Allergy (Copenhagen), 2014-05, Vol.69 (5), p.602-616 |
| issn | 0105-4538 1398-9995 |
| language | eng |
| recordid | cdi_proquest_miscellaneous_1520362408 |
| source | Wiley Free Content; MEDLINE; Wiley Online Library Journals Frontfile Complete; Elektronische Zeitschriftenbibliothek - Frei zugängliche E-Journals |
| subjects | Allergies angioedema Angioedema - diagnosis Angioedema - drug therapy Angioedema - etiology Classification clinical immunology dermatology education Humans urticaria |
| title | Classification, diagnosis, and approach to treatment for angioedema: consensus report from the Hereditary Angioedema International Working Group |
| url | https://sfx.bib-bvb.de/sfx_tum?ctx_ver=Z39.88-2004&ctx_enc=info:ofi/enc:UTF-8&ctx_tim=2025-01-31T15%3A20%3A46IST&url_ver=Z39.88-2004&url_ctx_fmt=infofi/fmt:kev:mtx:ctx&rfr_id=info:sid/primo.exlibrisgroup.com:primo3-Article-proquest_pubme&rft_val_fmt=info:ofi/fmt:kev:mtx:journal&rft.genre=article&rft.atitle=Classification,%20diagnosis,%20and%20approach%20to%20treatment%20for%20angioedema:%20consensus%20report%20from%20the%20Hereditary%20Angioedema%20International%20Working%20Group&rft.jtitle=Allergy%20(Copenhagen)&rft.au=Cicardi,%20M.&rft.aucorp=HAWK%20under%20the%20patronage%20of%20EAACI%20(European%20Academy%20of%20Allergy%20and%20Clinical%20Immunology)&rft.date=2014-05&rft.volume=69&rft.issue=5&rft.spage=602&rft.epage=616&rft.pages=602-616&rft.issn=0105-4538&rft.eissn=1398-9995&rft_id=info:doi/10.1111/all.12380&rft_dat=%3Cproquest_pubme%3E3268309841%3C/proquest_pubme%3E%3Curl%3E%3C/url%3E&disable_directlink=true&sfx.directlink=off&sfx.report_link=0&rft_id=info:oai/&rft_pqid=1513282576&rft_id=info:pmid/24673465&rfr_iscdi=true |