Management of Intraocular Hypertension During Hemodialysis by Intravenous Glucose Administration
A 64-year-old woman with end-stage renal disease and retinopathy secondary to type 2 diabetes mellitus presented with recurrent episodes of left ocular pain and acute loss of visual acuity during hemodialysis. During these episodes, markedly elevated intraocular pressures were measured. Several loca...
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Veröffentlicht in: | American journal of kidney diseases 2014-03, Vol.63 (3), p.500-502 |
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creator | Saritas, Turgay, MD Koutsonas, Antonis, MD Walter, Peter, MD Floege, Jürgen, MD Krüger, Thilo, MD |
description | A 64-year-old woman with end-stage renal disease and retinopathy secondary to type 2 diabetes mellitus presented with recurrent episodes of left ocular pain and acute loss of visual acuity during hemodialysis. During these episodes, markedly elevated intraocular pressures were measured. Several local and systemic antiglaucoma drugs were administered without improvement of intraocular pressure, resulting in the necessity of a glaucoma drainage device (Ahmed valve). Due to a local infection, it had to be removed, after which intraocular pressure elevations recurred during hemodialysis. Assuming that intraocular changes in osmolality during hemodialysis caused the intraocular pressure increases, intradialytic administration of a 20% glucose solution (100 mL/h) was initiated. This completely abrogated the development of both intraocular pain and increases in intraocular pressure. |
doi_str_mv | 10.1053/j.ajkd.2013.08.034 |
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During these episodes, markedly elevated intraocular pressures were measured. Several local and systemic antiglaucoma drugs were administered without improvement of intraocular pressure, resulting in the necessity of a glaucoma drainage device (Ahmed valve). Due to a local infection, it had to be removed, after which intraocular pressure elevations recurred during hemodialysis. Assuming that intraocular changes in osmolality during hemodialysis caused the intraocular pressure increases, intradialytic administration of a 20% glucose solution (100 mL/h) was initiated. This completely abrogated the development of both intraocular pain and increases in intraocular pressure.</description><identifier>ISSN: 0272-6386</identifier><identifier>EISSN: 1523-6838</identifier><identifier>DOI: 10.1053/j.ajkd.2013.08.034</identifier><identifier>PMID: 24189474</identifier><language>eng</language><publisher>New York, NY: Elsevier Inc</publisher><subject>Arterial hypertension. Arterial hypotension ; Biological and medical sciences ; Blood and lymphatic vessels ; blood glucose ; Cardiology. Vascular system ; diabetes mellitus ; eye ; Female ; Follow-Up Studies ; Glucose - administration & dosage ; Hemodialysis ; Humans ; Injections, Intravenous ; intraocular pressure ; Intraocular Pressure - drug effects ; Kidney Failure, Chronic - complications ; Kidney Failure, Chronic - therapy ; Medical sciences ; Middle Aged ; Nephrology ; Nephrology. Urinary tract diseases ; Ocular Hypertension - drug therapy ; Ocular Hypertension - etiology ; Ocular Hypertension - physiopathology ; Renal Dialysis ; Sweetening Agents - administration & dosage</subject><ispartof>American journal of kidney diseases, 2014-03, Vol.63 (3), p.500-502</ispartof><rights>National Kidney Foundation, Inc.</rights><rights>2014 National Kidney Foundation, Inc.</rights><rights>2015 INIST-CNRS</rights><rights>Copyright © 2014 National Kidney Foundation, Inc. Published by Elsevier Inc. All rights reserved.</rights><lds50>peer_reviewed</lds50><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c441t-f5e06d407a0d29d799b6e1f2eae04e4560d98527d1d869fb072236e350b0fba23</citedby><cites>FETCH-LOGICAL-c441t-f5e06d407a0d29d799b6e1f2eae04e4560d98527d1d869fb072236e350b0fba23</cites></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><linktohtml>$$Uhttps://dx.doi.org/10.1053/j.ajkd.2013.08.034$$EHTML$$P50$$Gelsevier$$H</linktohtml><link.rule.ids>314,780,784,3548,27922,27923,45993</link.rule.ids><backlink>$$Uhttp://pascal-francis.inist.fr/vibad/index.php?action=getRecordDetail&idt=28383490$$DView record in Pascal Francis$$Hfree_for_read</backlink><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/24189474$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Saritas, Turgay, MD</creatorcontrib><creatorcontrib>Koutsonas, Antonis, MD</creatorcontrib><creatorcontrib>Walter, Peter, MD</creatorcontrib><creatorcontrib>Floege, Jürgen, MD</creatorcontrib><creatorcontrib>Krüger, Thilo, MD</creatorcontrib><title>Management of Intraocular Hypertension During Hemodialysis by Intravenous Glucose Administration</title><title>American journal of kidney diseases</title><addtitle>Am J Kidney Dis</addtitle><description>A 64-year-old woman with end-stage renal disease and retinopathy secondary to type 2 diabetes mellitus presented with recurrent episodes of left ocular pain and acute loss of visual acuity during hemodialysis. During these episodes, markedly elevated intraocular pressures were measured. Several local and systemic antiglaucoma drugs were administered without improvement of intraocular pressure, resulting in the necessity of a glaucoma drainage device (Ahmed valve). Due to a local infection, it had to be removed, after which intraocular pressure elevations recurred during hemodialysis. Assuming that intraocular changes in osmolality during hemodialysis caused the intraocular pressure increases, intradialytic administration of a 20% glucose solution (100 mL/h) was initiated. This completely abrogated the development of both intraocular pain and increases in intraocular pressure.</description><subject>Arterial hypertension. Arterial hypotension</subject><subject>Biological and medical sciences</subject><subject>Blood and lymphatic vessels</subject><subject>blood glucose</subject><subject>Cardiology. Vascular system</subject><subject>diabetes mellitus</subject><subject>eye</subject><subject>Female</subject><subject>Follow-Up Studies</subject><subject>Glucose - administration & dosage</subject><subject>Hemodialysis</subject><subject>Humans</subject><subject>Injections, Intravenous</subject><subject>intraocular pressure</subject><subject>Intraocular Pressure - drug effects</subject><subject>Kidney Failure, Chronic - complications</subject><subject>Kidney Failure, Chronic - therapy</subject><subject>Medical sciences</subject><subject>Middle Aged</subject><subject>Nephrology</subject><subject>Nephrology. Urinary tract diseases</subject><subject>Ocular Hypertension - drug therapy</subject><subject>Ocular Hypertension - etiology</subject><subject>Ocular Hypertension - physiopathology</subject><subject>Renal Dialysis</subject><subject>Sweetening Agents - administration & dosage</subject><issn>0272-6386</issn><issn>1523-6838</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2014</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNp9kk1v1DAQhi0EotvCH-CAckHikjD-iJNICKkqtFupiANwNo49qZwmzmInlfLvcbQLSBw4-eDnnRk9M4S8olBQKPm7vtD9gy0YUF5AXQAXT8iOloznsub1U7IDVrFc8lqekfMYewBouJTPyRkTtG5EJXbkx2ft9T2O6Ods6rJbPwc9mWXQIduvBwwz-ugmn31cgvP32R7HyTo9rNHFrF2P_CP6aYnZzbCYKWJ2aUfnXUwfc0q-IM86PUR8eXovyPfrT9-u9vndl5vbq8u73AhB57wrEaQVUGmwrLFV07QSacdQIwgUpQTb1CWrLLW1bLoWKsa4RF5CC12rGb8gb491D2H6uWCc1eiiwWHQHtN0ipZAa84Fg4SyI2rCFGPATh2CG3VYFQW1mVW92syqzayCWiWzKfT6VH9pR7R_Ir9VJuDNCdDR6KEL2hsX_3JpJVw0W_f3Rw6TjUeHQUXj0Bu0LqCZlZ3c_-f48E_cDEl36viAK8Z-WoJPnhVVkSlQX7cb2E6AcqAsqeO_ALKyrMA</recordid><startdate>20140301</startdate><enddate>20140301</enddate><creator>Saritas, Turgay, MD</creator><creator>Koutsonas, Antonis, MD</creator><creator>Walter, Peter, MD</creator><creator>Floege, Jürgen, MD</creator><creator>Krüger, Thilo, MD</creator><general>Elsevier Inc</general><general>Elsevier</general><scope>IQODW</scope><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>7X8</scope></search><sort><creationdate>20140301</creationdate><title>Management of Intraocular Hypertension During Hemodialysis by Intravenous Glucose Administration</title><author>Saritas, Turgay, MD ; Koutsonas, Antonis, MD ; Walter, Peter, MD ; Floege, Jürgen, MD ; Krüger, Thilo, MD</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c441t-f5e06d407a0d29d799b6e1f2eae04e4560d98527d1d869fb072236e350b0fba23</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2014</creationdate><topic>Arterial hypertension. Arterial hypotension</topic><topic>Biological and medical sciences</topic><topic>Blood and lymphatic vessels</topic><topic>blood glucose</topic><topic>Cardiology. Vascular system</topic><topic>diabetes mellitus</topic><topic>eye</topic><topic>Female</topic><topic>Follow-Up Studies</topic><topic>Glucose - administration & dosage</topic><topic>Hemodialysis</topic><topic>Humans</topic><topic>Injections, Intravenous</topic><topic>intraocular pressure</topic><topic>Intraocular Pressure - drug effects</topic><topic>Kidney Failure, Chronic - complications</topic><topic>Kidney Failure, Chronic - therapy</topic><topic>Medical sciences</topic><topic>Middle Aged</topic><topic>Nephrology</topic><topic>Nephrology. Urinary tract diseases</topic><topic>Ocular Hypertension - drug therapy</topic><topic>Ocular Hypertension - etiology</topic><topic>Ocular Hypertension - physiopathology</topic><topic>Renal Dialysis</topic><topic>Sweetening Agents - administration & dosage</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Saritas, Turgay, MD</creatorcontrib><creatorcontrib>Koutsonas, Antonis, MD</creatorcontrib><creatorcontrib>Walter, Peter, MD</creatorcontrib><creatorcontrib>Floege, Jürgen, MD</creatorcontrib><creatorcontrib>Krüger, Thilo, MD</creatorcontrib><collection>Pascal-Francis</collection><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>MEDLINE - Academic</collection><jtitle>American journal of kidney diseases</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Saritas, Turgay, MD</au><au>Koutsonas, Antonis, MD</au><au>Walter, Peter, MD</au><au>Floege, Jürgen, MD</au><au>Krüger, Thilo, MD</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Management of Intraocular Hypertension During Hemodialysis by Intravenous Glucose Administration</atitle><jtitle>American journal of kidney diseases</jtitle><addtitle>Am J Kidney Dis</addtitle><date>2014-03-01</date><risdate>2014</risdate><volume>63</volume><issue>3</issue><spage>500</spage><epage>502</epage><pages>500-502</pages><issn>0272-6386</issn><eissn>1523-6838</eissn><abstract>A 64-year-old woman with end-stage renal disease and retinopathy secondary to type 2 diabetes mellitus presented with recurrent episodes of left ocular pain and acute loss of visual acuity during hemodialysis. During these episodes, markedly elevated intraocular pressures were measured. Several local and systemic antiglaucoma drugs were administered without improvement of intraocular pressure, resulting in the necessity of a glaucoma drainage device (Ahmed valve). Due to a local infection, it had to be removed, after which intraocular pressure elevations recurred during hemodialysis. Assuming that intraocular changes in osmolality during hemodialysis caused the intraocular pressure increases, intradialytic administration of a 20% glucose solution (100 mL/h) was initiated. This completely abrogated the development of both intraocular pain and increases in intraocular pressure.</abstract><cop>New York, NY</cop><pub>Elsevier Inc</pub><pmid>24189474</pmid><doi>10.1053/j.ajkd.2013.08.034</doi><tpages>3</tpages></addata></record> |
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subjects | Arterial hypertension. Arterial hypotension Biological and medical sciences Blood and lymphatic vessels blood glucose Cardiology. Vascular system diabetes mellitus eye Female Follow-Up Studies Glucose - administration & dosage Hemodialysis Humans Injections, Intravenous intraocular pressure Intraocular Pressure - drug effects Kidney Failure, Chronic - complications Kidney Failure, Chronic - therapy Medical sciences Middle Aged Nephrology Nephrology. Urinary tract diseases Ocular Hypertension - drug therapy Ocular Hypertension - etiology Ocular Hypertension - physiopathology Renal Dialysis Sweetening Agents - administration & dosage |
title | Management of Intraocular Hypertension During Hemodialysis by Intravenous Glucose Administration |
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