CCL21 attenuates HSV-induced inflammation through up-regulation of CD8+ memory cells

Abstract CCR7 and its ligand, CCL21, are known to establish microenvironments for the initiation of immune responses in secondary lymphoid tissue. It has also been reported that CCR7 ligand gene-deleted mice have defects in lymphocyte homing. In addition, the injection of the CCR7 ligand was shown t...

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Veröffentlicht in:	Immunobiology (1979) 2013-04, Vol.218 (4), p.579-590
Hauptverfasser:	Choi, Bunsoon, Lim, Hyun Chul, Lee, Eun-So, Anower, A.K.M. Mostafa, Sohn, Seonghyang
Format:	Artikel
Sprache:	eng
Schlagworte:	Adult Advanced Basic Science Allergy and Immunology Animals Behcet Syndrome - genetics Behcet Syndrome - immunology Behcet Syndrome - pathology Behcet Syndrome - virology Behçet's disease CCL21 CCR7 CD8+ memory T cells CD8-Positive T-Lymphocytes - immunology CD8-Positive T-Lymphocytes - pathology Cercopithecus aethiops Chemokine CCL21 - genetics Chemokine CCL21 - immunology Disease Models, Animal Female Herpes Simplex - genetics Herpes Simplex - immunology Herpes Simplex - pathology Herpes Simplex - virology Herpes simplex virus-induced inflammation Herpesvirus 1, Human - immunology Humans Immunologic Memory Inflammation - genetics Inflammation - immunology Inflammation - pathology Inflammation - virology Male Mice Mice, Inbred ICR Middle Aged Mouse model Receptors, CCR7 - genetics Receptors, CCR7 - immunology Up-Regulation - immunology Vero Cells
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