Cyclic AMP promotes axon regeneration, lesion repair and neuronal survival in lampreys after spinal cord injury

Axon regeneration after spinal cord injury in mammals is inadequate to restore function, illustrating the need to design better strategies for improving outcomes. Increasing the levels of the second messenger cyclic adenosine monophosphate (cAMP) after spinal cord injury enhances axon regeneration a...

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Veröffentlicht in:	Experimental neurology 2013-12, Vol.250, p.31-42
Hauptverfasser:	Lau, Billy Y.B., Fogerson, Stephanie M., Walsh, Rylie B., Morgan, Jennifer R.
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Axon regeneration Axons - metabolism Axons - pathology Axotomy Biological and medical sciences Blotting, Western Cell Survival - physiology Cerebrospinal fluid. Meninges. Spinal cord Cyclic AMP - metabolism Dibutyryl cyclic AMP (db-cAMP) Disease Models, Animal Fluorescent Antibody Technique Injuries of the nervous system and the skull. Diseases due to physical agents Lampreys Medical sciences Microglia/macrophages Nerve Regeneration - physiology Nervous system (semeiology, syndromes) Neurology Neuroprotection Petromyzontidae Plasticity Spinal Cord Injuries - metabolism SV2 Synaptotagmin Traumas. Diseases due to physical agents
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