The Temporal Development of Mood, Cognitive, and Vegetative Symptoms in Recurrent SAD Episodes: A Test of the Dual Vulnerability Hypothesis
Recent literature on seasonal affective disorder (SAD) has begun to focus on diathesis–stress models, including Young and colleagues’ dual vulnerability hypothesis. The dual vulnerability hypothesis posits that individuals must possess both a biological vulnerability to developing vegetative symptom...
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description | Recent literature on seasonal affective disorder (SAD) has begun to focus on diathesis–stress models, including Young and colleagues’ dual vulnerability hypothesis. The dual vulnerability hypothesis posits that individuals must possess both a biological vulnerability to developing vegetative symptoms and a psychological vulnerability to developing mood symptoms in order to develop SAD episodes. However, few studies have directly tested this model until very recently. Research has demonstrated a temporal relation between mood and vegetative symptoms, with vegetative symptoms having an earlier onset than mood symptoms supporting the idea that separate factors related to the two symptom clusters exist. The current study represents a longitudinal assessment of vegetative and mood symptoms, as well as cognitive factors (i.e., rumination, automatic thoughts) that may represent part of the psychological vulnerability shared by SAD sufferers. Furthermore, the present study represents only the second to assess state levels of cognitive factors that may impact recurrent SAD episode severity. Fifty-one individuals participated in the study across two groups, individuals with a history of SAD, and with no history of depression or SADs. Findings supported the dual vulnerability hypothesis, with an early vegetative symptom onset than mood symptom evident for the individuals with a history of SAD. Participants with a history of SAD also reported more ruminative responses and negative automatic thoughts about the seasons. Findings are generally supportive of Young and colleagues’ dual vulnerability hypothesis and directions for future research are suggested. |
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The dual vulnerability hypothesis posits that individuals must possess both a biological vulnerability to developing vegetative symptoms and a psychological vulnerability to developing mood symptoms in order to develop SAD episodes. However, few studies have directly tested this model until very recently. Research has demonstrated a temporal relation between mood and vegetative symptoms, with vegetative symptoms having an earlier onset than mood symptoms supporting the idea that separate factors related to the two symptom clusters exist. The current study represents a longitudinal assessment of vegetative and mood symptoms, as well as cognitive factors (i.e., rumination, automatic thoughts) that may represent part of the psychological vulnerability shared by SAD sufferers. Furthermore, the present study represents only the second to assess state levels of cognitive factors that may impact recurrent SAD episode severity. Fifty-one individuals participated in the study across two groups, individuals with a history of SAD, and with no history of depression or SADs. Findings supported the dual vulnerability hypothesis, with an early vegetative symptom onset than mood symptom evident for the individuals with a history of SAD. Participants with a history of SAD also reported more ruminative responses and negative automatic thoughts about the seasons. 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Fifty-one individuals participated in the study across two groups, individuals with a history of SAD, and with no history of depression or SADs. Findings supported the dual vulnerability hypothesis, with an early vegetative symptom onset than mood symptom evident for the individuals with a history of SAD. Participants with a history of SAD also reported more ruminative responses and negative automatic thoughts about the seasons. 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Psychiatry</topic><topic>Quality of Life Research</topic><topic>Seasonal affective disorder</topic><topic>Seasonal variations</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Whitcomb-Smith, Stacy</creatorcontrib><creatorcontrib>Sigmon, Sandra T.</creatorcontrib><creatorcontrib>Martinson, Amber</creatorcontrib><creatorcontrib>Young, Michael</creatorcontrib><creatorcontrib>Craner, Julia</creatorcontrib><creatorcontrib>Boulard, Nina</creatorcontrib><collection>Pascal-Francis</collection><collection>CrossRef</collection><collection>ProQuest Social Sciences Premium Collection</collection><collection>ProQuest Central (Corporate)</collection><collection>Neurosciences Abstracts</collection><collection>ProQuest Central (purchase pre-March 2016)</collection><collection>Psychology Database (Alumni)</collection><collection>Social Science Database (Alumni Edition)</collection><collection>ProQuest Pharma Collection</collection><collection>Hospital Premium Collection</collection><collection>Hospital Premium Collection (Alumni Edition)</collection><collection>ProQuest Central (Alumni) (purchase pre-March 2016)</collection><collection>Research Library (Alumni Edition)</collection><collection>ProQuest Central (Alumni Edition)</collection><collection>ProQuest Central UK/Ireland</collection><collection>Social Science Premium Collection</collection><collection>ProQuest Central Essentials</collection><collection>ProQuest Central</collection><collection>ProQuest One Community College</collection><collection>ProQuest Central Korea</collection><collection>Health Research Premium Collection</collection><collection>Health Research Premium Collection (Alumni)</collection><collection>ProQuest Central Student</collection><collection>Research Library Prep</collection><collection>Psychology Database</collection><collection>Research Library</collection><collection>Social Science Database</collection><collection>Research Library (Corporate)</collection><collection>ProQuest One Academic Eastern Edition (DO NOT USE)</collection><collection>ProQuest One Academic</collection><collection>ProQuest One Academic UKI Edition</collection><collection>ProQuest Central China</collection><collection>ProQuest One Psychology</collection><collection>ProQuest Central Basic</collection><jtitle>Cognitive therapy and research</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Whitcomb-Smith, Stacy</au><au>Sigmon, Sandra T.</au><au>Martinson, Amber</au><au>Young, Michael</au><au>Craner, Julia</au><au>Boulard, Nina</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>The Temporal Development of Mood, Cognitive, and Vegetative Symptoms in Recurrent SAD Episodes: A Test of the Dual Vulnerability Hypothesis</atitle><jtitle>Cognitive therapy and research</jtitle><stitle>Cogn Ther Res</stitle><date>2014-02-01</date><risdate>2014</risdate><volume>38</volume><issue>1</issue><spage>43</spage><epage>54</epage><pages>43-54</pages><issn>0147-5916</issn><eissn>1573-2819</eissn><coden>CTHRD8</coden><abstract>Recent literature on seasonal affective disorder (SAD) has begun to focus on diathesis–stress models, including Young and colleagues’ dual vulnerability hypothesis. The dual vulnerability hypothesis posits that individuals must possess both a biological vulnerability to developing vegetative symptoms and a psychological vulnerability to developing mood symptoms in order to develop SAD episodes. However, few studies have directly tested this model until very recently. Research has demonstrated a temporal relation between mood and vegetative symptoms, with vegetative symptoms having an earlier onset than mood symptoms supporting the idea that separate factors related to the two symptom clusters exist. The current study represents a longitudinal assessment of vegetative and mood symptoms, as well as cognitive factors (i.e., rumination, automatic thoughts) that may represent part of the psychological vulnerability shared by SAD sufferers. Furthermore, the present study represents only the second to assess state levels of cognitive factors that may impact recurrent SAD episode severity. Fifty-one individuals participated in the study across two groups, individuals with a history of SAD, and with no history of depression or SADs. Findings supported the dual vulnerability hypothesis, with an early vegetative symptom onset than mood symptom evident for the individuals with a history of SAD. Participants with a history of SAD also reported more ruminative responses and negative automatic thoughts about the seasons. Findings are generally supportive of Young and colleagues’ dual vulnerability hypothesis and directions for future research are suggested.</abstract><cop>Boston</cop><pub>Springer US</pub><doi>10.1007/s10608-013-9577-5</doi><tpages>12</tpages></addata></record> |
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subjects | Adult and adolescent clinical studies Biological and medical sciences Clinical Psychology Cognitive ability Cognitive Psychology Depression Emotions Hypotheses Medical sciences Medicine Medicine & Public Health Mental depression Mood disorders Original Article Psychology. Psychoanalysis. Psychiatry Psychopathology. Psychiatry Quality of Life Research Seasonal affective disorder Seasonal variations |
title | The Temporal Development of Mood, Cognitive, and Vegetative Symptoms in Recurrent SAD Episodes: A Test of the Dual Vulnerability Hypothesis |
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