Role of interleukin-1β in hypoxia-induced depression of glutamate uptake in retinal Müller cells
Background It is suggested that hypoxic–ischemic retinal diseases induce loss of retinal ganglion cells. Excess glutamate release is involved in these conditions. A predominant function of Müller cells is to regulate glutamate levels, but in these diseases the function is compromised. The present st...
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Veröffentlicht in: | Graefe's archive for clinical and experimental ophthalmology 2014, Vol.252 (1), p.51-58 |
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