AICAR inhibits oxygen consumption by intact skeletal muscle cells in culture

Activation of 5′ adenosine monophosphate-activated protein kinase (AMPK) with aminoimidazole carboxamide ribonucleotide (AICAR) increases skeletal muscle glucose uptake and fatty acid oxidation. The purpose of these experiments was to utilize AICAR to enhance palmitate consumption by mitochondria in...

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Veröffentlicht in:	Journal of physiology and biochemistry 2013-12, Vol.69 (4), p.909-917
Hauptverfasser:	Spangenburg, Espen E., Jackson, Kathryn C., Schuh, Rosemary A.
Format:	Artikel
Sprache:	eng
Schlagworte:	Aminoimidazole Carboxamide - analogs & derivatives Aminoimidazole Carboxamide - pharmacology AMP-Activated Protein Kinases - genetics AMP-Activated Protein Kinases - metabolism Animal Physiology Animals Biomedical and Life Sciences Biomedicine Cell Line Fatty Acids - metabolism Glucose - metabolism Human Physiology Mice Mitochondria, Muscle - drug effects Mitochondria, Muscle - metabolism Muscle Fibers, Skeletal - cytology Muscle Fibers, Skeletal - drug effects Muscle Fibers, Skeletal - metabolism Original Paper Oxidation-Reduction Oxygen - metabolism Oxygen Consumption - drug effects Oxygen Consumption - physiology Palmitic Acid - metabolism Phosphorylation Ribonucleotides - pharmacology
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