Central pontine myelinolysis following acute hypoglycemia

Here we present a case of a patient who developed CPM in the setting of acute hypoglycemia and Levofloxacin administration. 2 Case report A 46-year-old male with a history of alcohol abuse, pancreatitis, latent autoimmune diabetes of adulthood, and chronic kidney disease was transferred from an outside hospital after being found unresponsive. Reports of hypoglycemia-induced CPM/ODS are rare [3]; glucose deprivation can lead to cytotoxic edema secondary to failure of ion pumps in the cell membrane; this is reflected as a diffusion-weighted hyperintensity and corresponding decreased signal on ADC mapping as seen in our patient (Fig. 1).