Hepatitis B virus inhibits liver regeneration via epigenetic regulation of urokinase‐type plasminogen activator

Liver regeneration after liver damage caused by toxins and pathogens is critical for liver homeostasis. Retardation of liver proliferation was reported in hepatitis B virus (HBV) X protein (HBx)‐transgenic mice. However, the underlying mechanism of the HBx‐mediated disturbance of liver regeneration...

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Veröffentlicht in:	Hepatology (Baltimore, Md.) Md.), 2013-08, Vol.58 (2), p.762-776
Hauptverfasser:	Park, Eun‐Sook, Park, Yong Kwang, Shin, Chan Young, Park, Seung Hwa, Ahn, Sung Hyun, Kim, Doo Hyun, Lim, Keo‐Heun, Kwon, So Young, Kim, Kwang Pyo, Yang, Sung‐Il, Seong, Baik L., Kim, Kyun‐Hwan
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Cell Line Cell Proliferation Deoxyribonucleic acid Disease Models, Animal DNA DNA (Cytosine-5-)-Methyltransferases - physiology DNA, Viral - genetics Epigenesis, Genetic - physiology Epigenetics Hepatectomy Hepatitis B Hepatitis B - pathology Hepatitis B - physiopathology Hepatitis B virus - genetics Hepatitis B virus - physiology Hepatocyte Growth Factor - physiology Hepatocytes - pathology Hepatology In Vitro Techniques Liver Liver Regeneration - physiology Male Mice Mice, Inbred BALB C Mice, Transgenic Signal Transduction - physiology Trans-Activators - physiology Urokinase-Type Plasminogen Activator - physiology
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