Effect of warfarin treatment on thrombin activatable fibrinolysis inhibitor (TAFI) activation and TAFI‐mediated inhibition of fibrinolysis

Summary Background Severe clotting deficiencies are associated with enhanced in vitro fibrinolysis due to insufficient thrombin activatable fibrinolysis inhibitor (TAFI) activation. Because oral anticoagulant therapy (OAT) with warfarin causes a partial deficiency of vitamin K‐dependent factors, its...

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Veröffentlicht in:	Journal of thrombosis and haemostasis 2013-02, Vol.11 (2), p.315-324
Hauptverfasser:	Incampo, F., Carrieri, C., Galasso, R., Scaraggi, F. A., Di Serio, F., Woodhams, B., Semeraro, N., Colucci, M.
Format:	Artikel
Sprache:	eng
Schlagworte:	Administration, Oral Aged Aged, 80 and over alpha-2-Antiplasmin - metabolism Analysis of Variance Anticoagulants - administration & dosage Biomarkers - blood Carboxypeptidase B2 - blood Case-Control Studies clot resistance clotting deficiency Female Fibrin Clot Lysis Time Fibrinolysin - metabolism Fibrinolysis - drug effects Humans Male Middle Aged Nephelometry and Turbidimetry Peptide Fragments - blood Predictive Value of Tests Prothrombin TAFI Thrombelastography thrombin Thrombin - metabolism warfarin Warfarin - administration & dosage
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