Does laparoscopic gastric banding create hiatal hernias?

Abstract Background We hypothesized that laparoscopic adjustable gastric band (LAGB) placement might result in the development of a hiatal hernia (HH) over time. The objective of our study was to determine whether HHs develop after LAGB in the setting of a university hospital. Methods We retrospecti...

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Veröffentlicht in:Surgery for obesity and related diseases 2013, Vol.9 (1), p.48-52
Hauptverfasser: Azagury, Dan E., M.D, Varban, Oliver, M.D, Tavakkolizadeh, Ali, M.D, Robinson, Malcolm K., M.D, Vernon, Ashley H., M.D, Lautz, David B., M.D
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container_end_page 52
container_issue 1
container_start_page 48
container_title Surgery for obesity and related diseases
container_volume 9
creator Azagury, Dan E., M.D
Varban, Oliver, M.D
Tavakkolizadeh, Ali, M.D
Robinson, Malcolm K., M.D
Vernon, Ashley H., M.D
Lautz, David B., M.D
description Abstract Background We hypothesized that laparoscopic adjustable gastric band (LAGB) placement might result in the development of a hiatal hernia (HH) over time. The objective of our study was to determine whether HHs develop after LAGB in the setting of a university hospital. Methods We retrospectively reviewed all outcomes for consecutive LAGB patients in our institutional, longitudinal prospective bariatric surgical database to identify those patients without evidence of a HH at LAGB placement, who subsequently underwent delayed HH repair. Results From 2005 to 2009, 695 gastric bands were implanted. Twelve patients (1.72%) were identified who had no radiographic or intraoperative evidence of a HH at LAGB placement and who subsequently underwent HH repair at re-exploration. Patients presented 18 ± 10 months after band placement. Of these patients, 75% presented with gastroesophageal reflux disease or food intolerance (50% with gastroesophageal reflux disease alone). Also, 2 presented with acute pain due to band slippage and 1 with chronic pain and vomiting. In 50% of the patients, revision procedures detected the HH at operation despite negative preoperative studies. Conclusion In our series, a significant HH developed in 1.7% of LAGB patients who had no clinically identifiable HH at LAGB placement. Persistent dysphagia after band deflation requires careful inspection of the hiatus during surgical revision, even in the absence of radiologic depiction of HH, and might represent an underlying etiology of LAGB dysfunction. This complication, along with esophageal dilation and annular pouch dilation, might represent a constellation of conditions with a common etiology. From the results of our small series, we raise the question of the existence of chronic backpressure created by LAGB restriction and accounting for these complications.
doi_str_mv 10.1016/j.soard.2011.07.015
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The objective of our study was to determine whether HHs develop after LAGB in the setting of a university hospital. Methods We retrospectively reviewed all outcomes for consecutive LAGB patients in our institutional, longitudinal prospective bariatric surgical database to identify those patients without evidence of a HH at LAGB placement, who subsequently underwent delayed HH repair. Results From 2005 to 2009, 695 gastric bands were implanted. Twelve patients (1.72%) were identified who had no radiographic or intraoperative evidence of a HH at LAGB placement and who subsequently underwent HH repair at re-exploration. Patients presented 18 ± 10 months after band placement. Of these patients, 75% presented with gastroesophageal reflux disease or food intolerance (50% with gastroesophageal reflux disease alone). Also, 2 presented with acute pain due to band slippage and 1 with chronic pain and vomiting. In 50% of the patients, revision procedures detected the HH at operation despite negative preoperative studies. Conclusion In our series, a significant HH developed in 1.7% of LAGB patients who had no clinically identifiable HH at LAGB placement. Persistent dysphagia after band deflation requires careful inspection of the hiatus during surgical revision, even in the absence of radiologic depiction of HH, and might represent an underlying etiology of LAGB dysfunction. This complication, along with esophageal dilation and annular pouch dilation, might represent a constellation of conditions with a common etiology. From the results of our small series, we raise the question of the existence of chronic backpressure created by LAGB restriction and accounting for these complications.</description><identifier>ISSN: 1550-7289</identifier><identifier>EISSN: 1878-7533</identifier><identifier>DOI: 10.1016/j.soard.2011.07.015</identifier><identifier>PMID: 21925963</identifier><language>eng</language><publisher>United States: Elsevier Inc</publisher><subject>Complications ; Esophageal dilation ; Food Hypersensitivity - etiology ; Gastroenterology and Hepatology ; Gastroesophageal Reflux - etiology ; Gastroplasty - adverse effects ; Hernia, Hiatal - etiology ; Hernia, Hiatal - surgery ; Hiatal hernia ; Humans ; Laparoscopic adjustable gastric banding ; Laparoscopy - adverse effects ; Obesity, Morbid - surgery ; Pouch dilation ; Prospective Studies ; Reoperation ; Retrospective Studies ; Revision ; Surgery ; Weight Loss</subject><ispartof>Surgery for obesity and related diseases, 2013, Vol.9 (1), p.48-52</ispartof><rights>2013</rights><rights>Copyright © 2013. 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The objective of our study was to determine whether HHs develop after LAGB in the setting of a university hospital. Methods We retrospectively reviewed all outcomes for consecutive LAGB patients in our institutional, longitudinal prospective bariatric surgical database to identify those patients without evidence of a HH at LAGB placement, who subsequently underwent delayed HH repair. Results From 2005 to 2009, 695 gastric bands were implanted. Twelve patients (1.72%) were identified who had no radiographic or intraoperative evidence of a HH at LAGB placement and who subsequently underwent HH repair at re-exploration. Patients presented 18 ± 10 months after band placement. Of these patients, 75% presented with gastroesophageal reflux disease or food intolerance (50% with gastroesophageal reflux disease alone). Also, 2 presented with acute pain due to band slippage and 1 with chronic pain and vomiting. In 50% of the patients, revision procedures detected the HH at operation despite negative preoperative studies. Conclusion In our series, a significant HH developed in 1.7% of LAGB patients who had no clinically identifiable HH at LAGB placement. Persistent dysphagia after band deflation requires careful inspection of the hiatus during surgical revision, even in the absence of radiologic depiction of HH, and might represent an underlying etiology of LAGB dysfunction. This complication, along with esophageal dilation and annular pouch dilation, might represent a constellation of conditions with a common etiology. 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The objective of our study was to determine whether HHs develop after LAGB in the setting of a university hospital. Methods We retrospectively reviewed all outcomes for consecutive LAGB patients in our institutional, longitudinal prospective bariatric surgical database to identify those patients without evidence of a HH at LAGB placement, who subsequently underwent delayed HH repair. Results From 2005 to 2009, 695 gastric bands were implanted. Twelve patients (1.72%) were identified who had no radiographic or intraoperative evidence of a HH at LAGB placement and who subsequently underwent HH repair at re-exploration. Patients presented 18 ± 10 months after band placement. Of these patients, 75% presented with gastroesophageal reflux disease or food intolerance (50% with gastroesophageal reflux disease alone). Also, 2 presented with acute pain due to band slippage and 1 with chronic pain and vomiting. In 50% of the patients, revision procedures detected the HH at operation despite negative preoperative studies. Conclusion In our series, a significant HH developed in 1.7% of LAGB patients who had no clinically identifiable HH at LAGB placement. Persistent dysphagia after band deflation requires careful inspection of the hiatus during surgical revision, even in the absence of radiologic depiction of HH, and might represent an underlying etiology of LAGB dysfunction. This complication, along with esophageal dilation and annular pouch dilation, might represent a constellation of conditions with a common etiology. From the results of our small series, we raise the question of the existence of chronic backpressure created by LAGB restriction and accounting for these complications.</abstract><cop>United States</cop><pub>Elsevier Inc</pub><pmid>21925963</pmid><doi>10.1016/j.soard.2011.07.015</doi><tpages>5</tpages></addata></record>
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subjects Complications
Esophageal dilation
Food Hypersensitivity - etiology
Gastroenterology and Hepatology
Gastroesophageal Reflux - etiology
Gastroplasty - adverse effects
Hernia, Hiatal - etiology
Hernia, Hiatal - surgery
Hiatal hernia
Humans
Laparoscopic adjustable gastric banding
Laparoscopy - adverse effects
Obesity, Morbid - surgery
Pouch dilation
Prospective Studies
Reoperation
Retrospective Studies
Revision
Surgery
Weight Loss
title Does laparoscopic gastric banding create hiatal hernias?
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