Alteration in the Apoptosis Process of Rat Esophageal Epithelium with Hyperproliferation of Indigenous Bacteria under a Physiological Condition

The apoptosis process in rat esophageal epithelium was investigated using enzyme-immunohistochemistry and transmission electron microscopy. As a result, Fas and Fas-L were expressed in the epithelial cell membrane and cytoplasm from the stratum spinosum (SS) to the stratum granulosum (SG). No TNF-R1...

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Veröffentlicht in:	Journal of Veterinary Medical Science 2012, Vol.74(5), pp.597-605
Hauptverfasser:	UDAYANGA, Kankanam Gamage Sanath, YAMAMOTO, Kyoji, MIYATA, Hidenori, YOKOO, Yuh, MANTANI, Youhei, TAKAHARA, Ei-ichrou, KAWANO, Junichi, YOKOYAMA, Toshifumi, HOSHI, Nobuhiko, KITAGAWA, Hiroshi
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals apoptosis Apoptotic Protease-Activating Factor 1 - genetics Apoptotic Protease-Activating Factor 1 - metabolism Bacteria - growth & development bcl-2 Homologous Antagonist-Killer Protein - genetics bcl-2 Homologous Antagonist-Killer Protein - metabolism bcl-2-Associated X Protein - genetics bcl-2-Associated X Protein - metabolism BH3 Interacting Domain Death Agonist Protein - genetics BH3 Interacting Domain Death Agonist Protein - metabolism Caspase 9 - genetics Caspase 9 - metabolism Cell Proliferation Cytochromes c - genetics Cytochromes c - metabolism Epithelial Cells - cytology Epithelial Cells - microbiology Epithelial Cells - physiology Epithelium - microbiology esophageal epithelium Esophagus - microbiology Fas Ligand Protein - genetics Fas Ligand Protein - metabolism fas Receptor - genetics fas Receptor - metabolism Gene Expression Regulation immunohistochemistry indigenous bacteria Male rat Rats Rats, Wistar Receptors, Tumor Necrosis Factor - genetics Receptors, Tumor Necrosis Factor - metabolism Tumor Necrosis Factor-alpha - genetics Tumor Necrosis Factor-alpha - metabolism X-Linked Inhibitor of Apoptosis Protein - genetics X-Linked Inhibitor of Apoptosis Protein - metabolism
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